One dominant source of toxins for persons experiencing neurological challenges is polluted drinking water. One option many people embrace is to filter the water that they drink. This is a marked improvement over drinking unfiltered tap water. But what about distilled water? I this video I offer a comparison of distilled vs filtered water.

My honest confession is that I had never considered distilled water until a few months ago when it was recommended by our naturopath doctor. My initial reaction was to wonder whether bothering to distill water every day was really worth the effort.

What is Distilled Water?

Distilled water involves boiling it into steam, then condensing the steam back to a liquid. This removes toxins, bacteria, chemicals and microbes. It does take 5-6 hours to boil a gallon of water to make distilled water. Is the bother worth it?

As I point out in the video, I am shocked every day at the residue of gunk, sludge and goop that remains in the container after the gallon of water is distilled. Oh my. I have been putting that sludge into my body all this time (which has been a few decades).

Admittedly some of the sludge that remains consists of beneficial substances like minerals. But most of the gunk is toxic. I would prefer to get the minerals my body needs from eating nutritious food rather than drinking water that is laced with toxins.

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery ®
Road to Recovery from Parkinsons Disease

What are two simple suggestions to support recovery from Parkinsons?The vast number of natural therapies documented by Parkinsons Recovery that support recovery is indeed overwhelming. So many choices! Which ones offer the greatest possibility of relief for you?

The challenge for everyone is that no option – whether a prescription medicine, surgery or natural treatment helps everyone celebrate recovery. Most people celebrate relief from symptoms for many of the options but not everyone. It comes down to the challenge of deciding which treatment options are most likely to benefit you.

Two Simple Suggestions to Support Recovery

I asked Dr. Bruce Fife, ND, to offer two (and only two) suggestions among the many that are available that he suggests would be most likely to benefit the most people. Here is his unexpected answer:

Might I add that the cost of these two suggestions is minimal relative to the anticipated benefit. I say go for it!

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery®
https://www.parkinsonsrecovery.com
Road to Recovery from Parkinsons Disease

Studies published in 2026 pinpoint the value of a curumin treatment for Parkinsons.

What is Curcumin?

Curcumin is a spice used often in Indian, Asian and Middle Eastern cuisines for its earthy earthy flavor. Added often to curries, stews, rice and marinades. It is also widely used in health-focused cooking for its anti-inflammatory properties. 

It is actually an extract from Turmeric which is also a flavorful spice that offers gentle detoxes when added to food. Turmeric contains a variety compounds.  Curcumin is one of those compounds with health benefits because of its antioxidant and anti-inflammatory properties. Turmeric contains only up to 8% curcumin which makes curcumin supplements and spices far more concentrated than Tumeric. 

I received the following email reporting positive results from using curcumin to address dementia challenges.

 

Studies Published in 2026 Finding Benefits of Curcumin Treatment for Parkinsons

Front Pharmacol. 2026 Mar 9:17:1779921. Pharmacological effects and mechanisms of curcumin in animal models of Parkinson’s disease: a systematic review and meta-analysis

Abstract

Background: Curcumin has been demonstrated to possess promising neuroprotective potential in Parkinson’s disease; however, its overall effects remain inconclusive, and its multiple mechanisms of action have not been systematically summarized.

Objective: This systematic review and meta-analysis aimed to evaluate the pharmacological effects of curcumin in animal models of Parkinson’s disease and to investigate its potential mechanisms involving antioxidant, anti-inflammatory, and neuroprotective effects, thereby providing a theoretical basis for its potential clinical application in Parkinson’s disease.

Methods: A comprehensive search of four databases (EMBASE, PubMed, Web of Science, and the Cochrane Library) up to August 2025 identified 31 eligible studies involving a total of 552 animals. Methodological quality was assessed using the SYRCLE risk of bias tool. Standardized mean differences (SMDs) were calculated to evaluate the effects of curcumin on motor function, neurochemistry, inflammation, and oxidative stress in animal models of Parkinson’s disease.

Results: The results demonstrated that curcumin intervention improved motor function in animal models of Parkinson’s disease, as evidenced by increased locomotor distance in the open field test (SMD = 1.25) and elevated mean velocity (SMD = 1.42), prolonged latency to fall in the rotarod test (SMD = 2.49), shortened descent time in the pole test (SMD = -1.16), and reduced traversal time on the balance beam (SMD = -2.27). Curcumin exhibited neuroprotective effects through increasing the number of tyrosine hydroxylase-positive cells (SMD = 2.12), maintaining dopamine levels (SMD = 4.11), and elevating 3,4-dihydroxyphenylacetic acid concentrations (SMD = 3.15). Regarding anti-inflammatory effects, curcumin significantly reduced multiple inflammatory markers, including interleukin-6 (SMD = -4.73), interleukin-1? (SMD = -3.30), tumor necrosis factor-? (SMD = -3.19), and nitric oxide (SMD = -4.91). With respect to antioxidant activity, curcumin significantly reduced malondialdehyde levels (SMD = -4.69) while increasing the activities of superoxide dismutase (SMD = 3.90), glutathione (SMD = 2.08), and catalase (SMD = 2.00).

Conclusion: Curcumin demonstrates significant neuroprotective effects in Parkinson’s disease animal models, improving motor deficits and neuronal integrity likely through multi-target mechanisms involving anti-inflammatory and antioxidant pathways.

******

Drug Des Devel Ther. 2026 Jan 22:20:566113. Therapeutic Efficacy of Curcumin Nanoparticles in Parkinson’s Disease: An Integrated Analysis of Network Pharmacology, Experimental Validation, and Gut Microbiota

Abstract

Background: Parkinson’s disease (PD) ranks as the second most common neurodegenerative condition globally, with a rising occurrence alongside the aging demographic. Previous studies have demonstrated that curcumin monomers can alleviate PD symptoms and slow disease progression, whereas nano-drug delivery systems significantly improve their bioavailability. Notably, gut microbiota imbalance significantly contributes to the onset and advancement of PD; however, the mechanisms by which curcumin nanoparticles produce their therapeutic impact on PD are not yet well understood.

Materials and methods: Initially, network pharmacology was tapped to forecast the probable targets and signaling routes for curcumin in combating Parkinson’s Disease. Later on, molecular docking techniques and molecular dynamics experiments were utilized to substantiate its binding effectiveness. Curcumin nanoparticles were synthesized via the emulsion-solvent evaporation technique. After establishing PD models, multidimensional validation was performed using behavioral experiments, serological assays, Western blotting, hematoxylin-eosin staining, and 16S ribosomal RNA (16S rRNA) sequencing.

Results: Network pharmacology analysis revealed that curcumin acts through multiple targets and pathways. In vivo experiments demonstrated that curcumin nanoparticles enhanced motor capabilities in Parkinson’s disease mice, bolstered antioxidant enzyme levels, alleviated oxidative stress, and inhibited neuronal apoptosis via the Akt signaling pathway. Histopathological analysis showed significant improvements in the number and arrangement of neurons in the hippocampal dentate gyrus (DG) region. Furthermore, remodeling of the gut microbiota and metabolic regulation alleviated neuroinflammation and enhanced neuroprotection.

Conclusion: Curcumin nanoparticles inhibit neuronal apoptosis by activating the Akt signaling pathway and, while also remodeling the gut microbiota microenvironment, collectively ameliorate PD pathology. This study provides pharmacological evidence that curcumin nanoparticle therapy mediates its therapeutic actions through multiple mechanisms, and identifies potential therapeutic targets for PD treatment.

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery ®
Road to Recovery from Parkinsons Disease

Posted below is the fascinating article – Parkinson’s: A BioAcoustic Perspective – written by Sharry Edwards from Sound Health Options. The article presents findings from the BioAcoustic analysis for three individuals diagnosed with Parkinsons. This analysis reveals three very different causes.

Sharry’s research over the past several decades has identified more than a set of three factors that cause symptoms of Parkinsons. The Sound Health Options list of causes is quite extensive indeed. It includes among others …

• Allergies – especially to milk protein and gluten
• Lyme disease
• Peripheral neuropathy due to the measles virus
• Tetanus
• Inability to use glycine
• Lack of glutathione
• Stiff man syndroms
• Lack of receptors for some amino acids
• Trauma
• Emotional

Sharry uses analysis of voice profiles to determine causes. For more information about her revolutionary and groundbreaking approach and to obtain an anlysis of your voice profile visit:

https://www.soundhealthoptions.com

Or simply call her office which is located in Albany Ohio: 740-698-9119

Below is the article she recently published:

Parkinson’s: A BioAcoustic Perspective: Breaking the Sound Barrier of Parkinson’s Disease by Sharry Edwards

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Abstract

Parkinson’s disease is traditionally defined as a neurodegenerative disorder characterized by dopamine depletion and motor dysfunction. However, emerging evidence—supported by BioAcoustic vocal analysis—suggests that Parkinson’s may not be a singular disease entity, but rather a collection of distinct biological breakdown patterns that converge into similar symptoms.

This paper presents three case-based profiles demonstrating fundamentally different underlying mechanisms of Parkinson’s, as identified through frequency-based vocal analysis. These findings suggest a paradigm shift: Parkinson’s may be better understood as a pattern of expression rather than a single disease, requiring individualized identification and intervention.

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Introduction: The Limits of a Single Diagnosis

Parkinson’s disease has long been associated with:

• Loss of dopaminergic neurons
• Tremors, rigidity, and slowed movement
• Progressive neurological decline

While these symptoms are consistent, the cause is not.

Patients with identical diagnoses often respond differently to the same treatments. This inconsistency suggests that Parkinson’s may arise from multiple biological pathways—each requiring a different approach.

BioAcoustic Biology proposes that the human voice is a real-time representation of physiological function, reflecting biochemical, neurological, and structural changes. Through vocal analysis, it becomes possible to identify which system is breaking down—not just that a breakdown exists.

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Methodology: Listening to Biology

Using BioAcoustic vocal profiling, three individuals diagnosed with Parkinson’s disease were evaluated. Each voice sample was analyzed across multiple biological layers, including:

• Neurotransmitter function
• Immune signaling
• Detoxification pathways
• Structural and muscular integrity

Despite sharing a common diagnosis, each individual demonstrated a distinct underlying pattern of dysfunction.

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Case Study 1: The Gut–Immune–Neuroinflammation Pathway

Primary Features

• Intestinal permeability (“leaky gut”)
• Elevated inflammatory markers (Interleukin-17)
• Tetanus-related bacterial signaling
• Disruption of glutathione and glycine metabolism

Mechanism

In this case, the origin appears to begin in the gut:

Breakdown of the intestinal barrier ? immune activation ? systemic inflammation ? neuroinflammation ? motor symptoms

Interleukin-17, a pro-inflammatory cytokine, is known to promote neuroinflammation and disrupt the blood-brain barrier. Combined with impaired detoxification (glutathione dysfunction), this creates a biochemical environment that may damage dopaminergic neurons.

Clinical Implication

This type of Parkinson’s may be driven primarily by:

• Immune dysregulation
• Gut integrity failure
• Chronic inflammatory signaling

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Case Study 2- Thom: The Toxic–Oxidative Stress Pathway

Primary Features

• Severe systemic toxicity
• Widespread glutathione pathway disruption
• Oxidative stress overload
• Impaired cellular energy production

Mechanism

This profile reflects a breakdown in the body’s ability to detoxify:

Toxic burden ? glutathione depletion ? oxidative stress ? mitochondrial damage ? neuronal degeneration

Glutathione, a critical antioxidant, is central to neutralizing free radicals. In this case, multiple enzymes responsible for glutathione synthesis and utilization were compromised, indicating a system-wide failure of detoxification.

Clinical Implication

This type of Parkinson’s may be driven primarily by:

• Environmental or metabolic toxicity
• Antioxidant depletion
• Mitochondrial dysfunction

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Case Study 3- John: The Neurotransmitter–Structural Breakdown Pathway

Primary Features

• Acetylcholine receptor dysfunction
• Glycine receptor disruption
• Impaired dopaminergic neuron support (GDNF pathway)
• Muscle structure and nerve repair deficits

Mechanism

Here, the issue is not primarily immune or toxic, but neurological signaling and repair:

Neurotransmitter receptor dysfunction ? impaired signaling ? reduced neuron support ? failed repair mechanisms ? motor dysfunction

The GDNF (Glial Cell Line-Derived Neurotrophic Factor) pathway, essential for the survival of dopaminergic neurons, appears compromised. Simultaneously, structural proteins responsible for muscle coordination and nerve integrity show dysfunction.

Clinical Implication

This type of Parkinson’s may be driven primarily by:

• Neurotransmitter imbalance
• Failure of neuronal support systems
• Structural degeneration of muscle and nerve tissue

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Comparative Analysis: One Disease, Three Origins

These three cases reveal a critical insight:

Pathway	Primary Driver	Key System
Gut–Immune	Inflammation (IL-17)	Immune + Digestive
Toxic–Oxidative	Glutathione failure	Detox + Mitochondria
Neurotransmitter–                   Structural	Signaling breakdown	Nervous + Muscular

Despite identical diagnoses, the root causes differ dramatically.

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Discussion: Rethinking Parkinson’s

The current medical model treats Parkinson’s as a uniform condition, often focusing on dopamine replacement. While this may address symptoms, it does not address cause.

These findings suggest:

• Parkinson’s is a multi-origin condition
• Symptoms are shared endpoints, not shared beginnings
• Effective intervention must be pathway-specific

BioAcoustic analysis offers a unique advantage: it identifies dysfunction at the level of pattern and frequency, before structural damage becomes irreversible.

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Conclusion: A New Model of Understanding

Parkinson’s disease may not be a single disease—but rather a collection of biological failures that sound the same.

If this is true, then:

• Diagnosis must evolve beyond symptom classification
• Treatment must become individualized
• Early detection must focus on functional change, not structural damage

The human voice may provide a key to this shift—offering a non-invasive, real-time window into the body’s internal state.

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Author Perspective

For over four decades, BioAcoustic Biology has explored the relationship between sound, structure, and physiology. The voice is not merely a tool for communication—it is a mathematical expression of the body’s current condition.

By learning to interpret this expression, we may move toward a future where disease is identified and addressed before it fully manifests.

COMPARATIVE SUMMARY (VERY IMPORTANT)

Mechanism	Client 1	Client 2	Client 3
Gut involvement	Strong
Immune / IL-17	Strong	Mild	Possible
Toxicity	Moderate	Severe	Secondary
Glutathione		(dominant)
Neurotransmitter receptors			Strong
Dopamine neuron support (GDNF)			Strong
Structural/muscle degeneration			Strong

“We won’t have a cure for diseases until we first have a cure for greed.”

                                                                         Dr. Sachin Patel

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery
Road to Recovery from Parkinsons Disease

What is the meaning of numbers on fruit stickers? Is that lemon you are thinking of buying at the grocery store really organic?

Numbers on fruit stickers at grocery stores have either 4 or 5 digits. If the fruit you are about to place into your grocery cart has a 4-digit code, might I suggest you leave the fruit on the grocery shelves. Four digit stickers mean that the fruit was grown using pesticides, fertilizers and other toxic chemicals.

If the code on your fruit is a 5-digit number, there are two possibilities. One is good and the other is not. If the 5-digit code begins with a 9, it shows that the produce was grown organically. Great, That is the preferred option.

If the code begins with an 8, it means that the fruit has been genetically modified. Best to pass on that option given widespread concerns about the dangers of genetically modified foods which have been banned in Europe. Over 19 countries, including France, Germany, and Italy have banned the cultivation of GMO crops

Now a caution is in order. Even if the fruit was grown organically – no harmful toxins applied – there still may be an accumulation of toxins from the soil.

The best option of all is to buy foods with no stickers from local farmers. Visit your local farmers market often. Befriend local farmers who grow their crops organically. You will enjoy the experience and best of all you will begin to feel better too.

A final note on the impact of stickers on food. The study below finds that young people do not make choices based on labeling whether a food was genetically modified or not. I recommend that everyone – young and old – take the labeling on food stickers into consideration when making purchase decisions.

Int J Environ Res Public Health. 2021 Feb 11;18(4):1761. GMO Food Labels Do Not Affect College Student Food Selection, Despite Negative Attitudes towards GMOs

Abstract

US Public Law 114-216 dictates that food producers in the United States of America will be required to label foods containing genetically modified organisms (GMOs) starting in 2022; however, there is little empirical evidence demonstrating how U.S. consumers would use food labels that indicate the presence or absence of GMOs.

The aim of this two-phase study was to determine how attitudes towards GMOs relate to food choices and how labels indicating the presence or absence of GMOs differentially impact choices among college students-the age group which values transparent food labeling more than any other. Participants (n = 434) made yes/no choices for each of 64 foods. In both phases of the study, participants were randomly assigned to seeing GMO Free labels, contains GMOs labels, or no GMO labels.

Across the two phases, 85% of participants reported believing that GMOs were at least somewhat dangerous to health (42% believed GMOs to be dangerous), yet in both studies, although eye-tracking data verified that participants attended to the GMO labels, these labels did not significantly affect food choices. Although college consumers may believe GMOs to be dangerous, their food choices do not reflect this belief.

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery ®
Road to Recovery from Parkinsons Disease

Make 2026 your best year ever by joining my one on one mentoring program. Imagine how differently you will feel after working together with me as we join together to fit together all the pieces of a successful recovery program.

Introducing my One on One  Mentorship Program – Your Personal Roadmap to Recovery from neurological challenges. To enroll in the program click on the registration link below.

One on One Mentoring with Robert

 My mentoring program is all about having the right plan of recovery customized specifically for your unique situation as you celebrate relief from Parkinsons with natural therapies and methods.

What is Included

1. Initial strategy session with me personally to flesh out the potential causes of your symptoms so we can identify therapies and treatments that can successfully address them. This is not some surface-level consultation. It is a deep dive into your specific history and situation. We evaluate what you have tried that did not succeed and what did help. We consider new options.  

2. Together we lay out a custom-crafted program of recovery created specifically for your situation.

3. With bimonthly hourly meetings (with a video type interface similar to Zoom or telephone/email contact), we review, update and revise your program of recovery. 

• We review your progress since our last session
• Identify and solve any roadblocks you have confronted.
• Refine the recovery program based on real-world results documented by research
• Help keep you on track with your program of recovery
• Support your progress with new therapeutic and treatment options

4. Unlimited email access to me personally

Why Do I Offer One on One Mentoring

First – After nearly 22 years of researching the causes of Parkinsons and the natural therapies that can reverse them,  I have reached a point where my greatest satisfaction in life comes from seeing more and more individuals transform their health.

Second – The individuals I have been mentoring over recent months have been making remarkable progress. This warms my heart and soothes my soul to know that the ongoing attention and support is making a difference.

To claim one of the five enrollments available visit the registration link below.
One on One Mentoring with Robert

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery®
https://www.parkinsonsrecovery.com

The answer turns on the presence of low sulfur levels that derail the success of any detox program. The body loses its ability to process toxins and medications. Sulfur is needed to synthesize glutathione which is well documented to be seriously deficient in persons diagnosed with Parkinsons.

Glutathione binds to toxins such as heavy metals like mercury and aluminum for extraction from the body. Deficient levels of both sulfur and glutathione increase free radicals and cause cellular damage. 

To summarize – sulfur is a critical mineral required for the body’s major detoxification pathways. It plays an essential role in processing and eliminating toxins, medications, hormones, and neurotransmitters. When availability of sulfur is deficient, any detox protocol will be sidelined.

Foods Sources of Sulfur

Recent Research that Explains Why Detoxes Flop

Front Nutr. 2025 Apr 9:12:1534000. Natural sulfur compounds in mental health and neurological disorders: insights from observational and intervention studies

Abstract

Over the years, the global disease burden of neurological disorders (NDs) and mental disorders (MDs) has significantly increased, making them one of the most critical concerns and challenges to human health. In pursuit of novel therapies against MD and ND, there has been a growing focus on nutrition and health.

Dietary sulfur, primarily derived from various natural sources, plays a crucial role in numerous physiological processes, including brain function. This review offers an overview of the chemical composition of several natural sources of the sulfur-rich substances such as isothiocyanates, sulforaphane, glutathione, taurine, sulfated polysaccharides, allyl sulfides, and sulfur-containing amino acids, all of which have neuroprotective properties.

A multitude of studies have documented that consuming foods that are high in sulfur enhances brain function by improving cognitive parameters and reduces the severity of neuropathology by exhibiting antioxidant and anti-inflammatory properties at the molecular level.

In addition, the growing role of natural sulfur compounds in repairing endothelial dysfunction, compromising blood-brain barrier and improving cerebral blood flow, are documented here.

This review covers the encouraging results of supplementing sulfur-rich diets in many animal models and clinical investigations, along with their molecular targets in MD, such as schizophrenia, depression, anxiety, bipolar disorder, and autism spectrum disorder, and ND, such as Alzheimer’s disease (AD), Parkinson’s disease (PD), Amyotrophic Lateral Sclerosis (ALS), and Multiple Sclerosis (MS).

The prospects of natural sulfur compounds show great promise as they have potential applications in nutraceuticals, medicines, and functional foods to enhance brain function and prevent diseases.

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery®
https://www.parkinsonsrecovery.com
robert@parkinsonsrecovery.com

Many individuals have problems with celebrating restful sleep at night because of tremors that keep them awake. What is the cause of night time tremors which keep people from getting a good night’s sleep?

Many people think the problem of night time tremors is simply a result of a dopamine deficiency. Researchers have recently identified a very different reason for troublesome tremors at night time that keep many people with Parkinsons from getting restful sleeping at night.

Low blood sugar levels at night time – a condition called hypoglycemic – contribute to a variety of symptoms including

• Tremors
• Nervousness
• Sweating
• Chills
• Irritability
• Fast heartbeat
• Confusion
• Headache

Note the overlap of the symptoms of hypoglycemic listed above and those of Parkinsons which include among others tremors, anxiety, irritability, cognitive dysfunction and depression. The synergetic effect of these symptoms result in tremors that keep people awake in the evening or wake them up throughout during the night.

Research Connecting Hypoglycemia with Parkinsons Symptoms

Neurol Neurochir Pol. 2025;59(6):570-575. Episodes of hypoglycemia in patients with Parkinson’s disease – results of two-week continuous glucose monitoring

Abstract

Aim of the study: Recent evidence suggests that Parkinson’s disease (PD) and glucose metabolism disorders may share some common dysregulation pathways. This study sought to determine the incidence of hypoglycemic episodes in PD patients and identify associated risk factors for these events.

Clinical rationale for the study: Hypoglycemia in PD, especially at night, is not well understood. This study quantified hypoglycemia in PD patients and identified autonomic dysfunction as a key risk factor, providing new insights for research into metabolic aspects of PD.

Material and methods: Thirty-five patients diagnosed with PD and 20 control subjects underwent a study consisting of a clinical and biometric assessment and two weeks of continuous glucose monitoring using the Freestyle Libre system to detect episodic hypoglycemia.

Results: Among PD patients, 18 (51.4%) had episodes of hypoglycemia, and 67.8% of the events occurred between 0:00 and 6:00 a.m. Compared to the control group, there were significantly more hypoglycemic episodes registered in PD patients (3.6/pt vs. 1.4/pt; p = 0.0006). Predictors of the occurrence of hypoglycemic episodes in the PD group were lower two-week average glycemic levels (OR: 0.85, p = 0.016), glycemic variability index (OR: 1.46, p = 0.02), and SCOPA-AUT scale score (OR: 1.23, p = 0.04).

Conclusions and clinical implications: Patients with PD are at higher risk of hypoglycemic episodes, especially at night and in the morning. Patients with autonomic dysfunction are at risk for hypoglycemic episodes. Recognizing hypoglycemic episodes can enhance the management of PD by enabling targeted interventions aimed at reducing glycemic variability.

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery ®
https://www.parkinsonsrecovery.com
Road to Recovery from Parkinsons Disease
https://www.parkinsonsdisease.me

Elderberry, which comes from the Sambucus tree, is a complex of plants native to most of Europe. Common names include elder, elderberry, black elder, European elder, European elderberry, and European black elderberry. Many people are unaware of the elderberry benefits for Parkinsons.

Rich in antioxidants like anthocyanins, Elderberry reduces inflammation and fights oxidative stress, both of which are associated with Parkinson’s symptoms.

In light of recent research on Elderberry to treat Parkinson’s symptoms, it is one of the supplements among others that is beneficial to treat Parkinsons symptoms and merits further consideration and discussion with your doctor. This would be especially the case if inflammation is one of the factors identified by you and your doctor that needs to be treated and addressed.

Elderberry is available as an over the counter supplement and does not require a prescription from a doctor.

Research on Elderberry Benefits for Parkinsons

Int J Mol Sci. 2024 Jul 18;25(14):7843. Genus Sambucus (Elderberry): Exploring Its Potential as a Functional Food Ingredient with Neuroprotective Properties Mediated by Antioxidant and Anti-Inflammatory Mechanisms

Abstract

The genus Sambucus, mainly Sambucus nigra, has emerged as a valuable source of bioactive compounds with potential neuroprotective properties. This review explores the antioxidant, anti-inflammatory, and neuroregenerative effects of Sambucus-derived compounds and their implications for brain health and cognitive function.

In vitro studies have demonstrated the ability of Sambucus extracts to mitigate oxidative stress, modulate inflammatory responses, and promote neural stem cell proliferation and differentiation.

In vivo studies using animal models of neurodegenerative diseases, such as Alzheimer’s and Parkinson’s, have shown that Sambucus compounds can improve cognitive function, motor performance, and neuronal survival while attenuating neuroinflammation and oxidative damage.

The neuroprotective effects of Sambucus are primarily attributed to its rich content of polyphenols, particularly anthocyanins, which exert their benefits through multiple mechanisms. These include the modulation of signaling pathways involved in inflammation, apoptosis, mitochondrial function, and oxidative stress.

This review provides a comprehensive overview of the current state of research on the neuroprotective potential of Sambucus and its derivatives, offering valuable insights for the development of dietary strategies to promote brain health and prevent age-related cognitive decline.

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery ®
Road to Recovery from Parkinsons Disease

Hypomimia or “facial masking” is a restriction in the ability of an individual to express their emotions with facial expressions. The individual has a frozen, blank stare, despite the fact that intense feelings may be experienced. Smile therapy has is shown by research to thaw a frozen face.

• Why in the world might this be the case?
• How can flexible movements in a persons face improve gait disturbances?

What is Smile Therapy?

Smile therapy consists of exercises to facilitate the ability of a person to express their emotions. Exercises encourage the active, voluntary movement of facial muscles to combat facial rigidity.

Smile training involves facial muscle exercises to improve gait speed and reduce slowness of movement. Exercises involve making “surprised” faces and performing “silly” fish-like faces to stretch the muscles around the mouth, eyes and forehead.

Research on the Connection between Smile Therapy and Gait Disturbances

Phys Ther Res. 2024;27(3):173-179. Effects of Smile Training on Gait Disturbance in Parkinson’s Disease Patient with Neuropsychiatric Symptoms: A Single Case Design

Abstract

Objective: To verify the efficacy of smile training in improving gait disturbances in patients with Parkinson’s disease (PD) exhibiting neuropsychiatric symptoms.

Methods: A single-case design with three intervention periods (B1, A1, and B2) was used. During periods B1 and B2, 10 min of smile training (facial muscles training and positive thinking training) was performed before the usual exercise therapy. During the A1 period, the participant received only the usual exercise therapy. During the intervention period, the Timed Up and Go test (TUG) was performed daily in both directions. Tau-U was calculated to determine the effect size of the TUG test time and the number of steps taken during each period.

Movement Disorder Society-Unified Parkinson’s Disease Rating Scale (MDS-UPDRS) Part III, Hospital Anxiety and Depression Scale (HADS), 10-meter walk at maximum speed, Berg Balance Scale, and Characterizing Freezing of Gait Questionnaire (C-FOGQ) were administered on the day before the start of the intervention and the last day of each period.

Results: Comparisons of A1 to B2, TUG time, and the number of steps taken on both turns revealed large reductions (Tau-U ?0.74, p <0.01). The 10-meter walk speed and MDS-UPDRS Part III bradykinesia scores improved, whereas the frequency of gait freezing on the C-FOGQ remained unchanged. The HADS scores did not show significant changes; however, the participant made more positive statements in his reflections.

Conclusion: Smile training may be an effective intervention for improving gait and other motor symptoms in patients with PD.

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery®
https://www.parkinsonsdisease.me

Treating anxiety with ashwagandha is a recommendation backed up by sound research evidence. Several recent review studies conclude the herbal remedy ashwagandha reduces anxiety, supports restful sleep and improves overall cognitive functioning.

What is Ashwagandha?

Ashwagandha root has been used in the traditional Ayurvedic medicine in India as an adaptogen, a compound that increases a person’s ability to become resilient to biological, physical or chemical stressors. Most of the commercial ashwagandha supplements contain extracts from the plant’s root. Some contain extracts from both the root and leaf.

My Shut Down Anxiety online course covers a variety of different ways to shut down anxiety, the key to celebrating relief from symptoms. Information and course registration is available at: Shut Down Anxiety. Enter the coupon code succeed to claim a 50% discount off of the course tuition.

Studies Recommend Treating Anxiety with Ashwagandha

Results from clinical trials suggest that ashwagandha reduces stress and anxiety. A 2021 systematic review of the research evidence identified seven studies that investigated the use of ashwagandha to treat stress and anxiety.

A total of 491 adults, all from India, with either self-reported high stress and anxiety or a diagnosed anxiety disorder were randomized to take ashwagandha or placebo for 6 to 8 weeks.

The more recent meta-analytic reviews of Ashwagandha studies listed below confirm results of the earlier 2021 review summarized above that conclude Ashwagandha has a beneficial impact on shutting down anxiety.

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Explore (NY). 2024 Nov-Dec;20(6):103062. Effects of Ashwagandha (Withania Somnifera) on stress and anxiety: A systematic review and meta-analysis

Abstract

Background: Ashwagandha (Withania somnifera) is an adaptogenic herb used to prevent and treat psychosomatic disorders. This systematic review and meta-analysis evaluated the effects and safety of Ashwagandha on psychosomatic functions related to stress and anxiety among patients.

Methods: A comprehensive search was conducted in MEDLINE, EMBASE, PubMed, PsychINFO, and the Cochrane Library for articles published from January 2000 to January 2022. Randomized controlled trials that examined the effects of Ashwagandha on stress and anxiety were included. Two authors independently extracted all relevant data from the included studies. Both subjective and objective measures of stress and anxiety were assessed as outcome variables.

Result: Nine randomized controlled trials involving 558 patients were eligible for this study. The findings of the meta-analysis showed a significant effect of Ashwagandha formulations on the Perceived Stress Scale (PSS) (MD = -4.72, 95 % CI = [-8.45 to -0.99]), Hamilton Anxiety Scale (HAS) (MD = -2.19, 95 % CI = [-3.83 to -0.55]), and serum cortisol levels (MD = -2.58, 95 % CI = [-4.99 to -0.16]) compared to the placebo group. Among the included studies, four reported mild to moderate adverse events.

Conclusion: The findings from the included studies indicate that Ashwagandha formulations have beneficial effects on stress and anxiety.

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Phytother Res. 2022 Nov;36(11):4115-4124. Does Ashwagandha supplementation have a beneficial effect on the management of anxiety and stress? A systematic review and meta-analysis of randomized controlled trials

Abstract

Background: Clinical trial studies revealed conflicting results on the effect of Ashwagandha extract on anxiety and stress. Therefore, we aimed to evaluate the effect of Ashwagandha supplementation on anxiety as well as stress.

Method: A systematic search was performed in PubMed/Medline, Scopus, and Google Scholar from inception until December 2021. We included randomized clinical trials (RCTs) that investigate the effect of Ashwagandha extract on anxiety and stress. The overall effect size was pooled by random-effects model and the standardized mean difference (SMD) and 95% confidence interval (CIs) for outcomes were applied.

Overall, 12 eligible papers with a total sample size of 1,002 participants and age range between 25 and 48 years were included in the current systematic review and meta-analysis.

Results: We found that Ashwagandha supplementation significantly reduced anxiety (SMD: -1.55, 95% CI: -2.37, -0.74; p = .005; I² = 93.8%) and stress level (SMD: -1.75; 95% CI: -2.29, -1.22; p = .005; I² = 83.1%) compared to the placebo. Additionally, the non-linear dose-response analysis indicated a favorable effect of Ashwagandha supplementation on anxiety until 12,000 mg/d and stress at dose of 300-600 mg/d.

Conclusion: The current systematic review and dose-response meta-analysis of RCTs revealed a beneficial effect in both stress and anxiety following Ashwagandha supplementation. 

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery ®
https://www.blog.parkinsonsrecovery.com
robert@parkinsonsrecovery.com

Inflammation is a core feature of Parkinson’s disease, so why not consider natural antioxidants as treatments for Parkinsons? Admittedly, this recommendation may likely be strange to many of you.

Most individuals first diagnosed with Parkinsons focus on dopamine as the primary culprit for their symptoms. This belief leads to the conclusion that the best treatment option is take medicines and supplements that infuse the body with L-dopa, a precursor of dopamine. No other treatment option need be even considered.

Given that studies consistently find inflammation is a core feature of Parkinson’s disease, a companion option is to consider also taking antioxidants as treatments for Parkinsons. Of course, antioxidants can be prescribed by your doctor. A variety of natural antioxidants do not require a prescription.

The following is a list of natural antioxidants that offer the opportunity to celebrate a relief from symptoms inflamed by inflammation:

• N-acetylcysteine
• sulforaphane
• alpha-lipoic acid
• L-carnitine
• ascorbic acid (Vitamin C)
• selenocompounds
• flavones
• zinc
• Vitamin E

Why not select a few of the above natural antioxidants to take on a regular basis to keep inflammation from overcoming the body’s ability to rejuvenate and regenerate?

Research on Antioxidants as Treatments for Parkinsons

Front Neurosci. 2024 Dec 5:18:1505153. Antioxidants in neuropsychiatric disorder prevention: neuroprotection, synaptic regulation, microglia modulation, and neurotrophic effect

Abstract

Oxidative stress, caused by an imbalance between the generation of reactive oxygen species (ROS) and the body’s intrinsic antioxidant defenses, plays a critical role in neurodegenerative diseases such as Alzheimer’s, Parkinson’s, and Huntington’s. Beyond these conditions, recent evidence indicates that dysregulated redox balance is implicated in neuropsychiatric disorders, including schizophrenia, major depressive disorder, and anxiety disorders.

Preclinical and clinical studies have demonstrated the potential of antioxidants, such as N-acetylcysteine, sulforaphane, alpha-lipoic acid, L-carnitine, ascorbic acid, selenocompounds, flavones and zinc, in alleviating neuropsychiatric symptoms by mitigating excitotoxicity, enhancing synaptic plasticity, reducing microglial overactivation and promoting synaptogenesis.

This review explores the role of oxidative stress in the pathogenesis of neuropsychiatric disorders. It provides an overview of the current evidence on antioxidant therapy’s pharmacological effects, as demonstrated in animal models and clinical studies.

Given the limitations and side effects of existing treatments for neuropsychiatric disorders, antioxidant therapy presents a promising, safer alternative. Further research is essential to deepen our understanding and investigate the clinical efficacy and mechanisms underlying these therapies.

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Adv Nutr. 2022 Oct 2;13(5):1493-1504. Dietary Antioxidants and Risk of Parkinson’s Disease: A Systematic Review and Dose-Response Meta-analysis of Observational Studies

Abstract

The aim of the current review was to explore the association between various dietary antioxidants and the risk of developing Parkinson’s disease (PD).

PubMed, Scopus, Web of Science, and Google Scholar were searched up to March 2021. Prospective, observational cohort studies, nested case-control, and case-control designs that investigated the association between antioxidants and PD risk were included. A random-effects model was used to pool the RRs. The certainty of the evidence was rated using the GRADE (Grading of Recommendations Assessment, Development, and Evaluations) scoring system. In addition, a dose-response relation was examined between antioxidant intake and PD risk.

Six prospective cohort studies and 2 nested case-control (total n = 448,737 with 4654 cases), as well as 6 case-control (1948 controls, 1273 cases) studies were eligible. The pooled RR was significantly lower for the highest compared with the lowest intake categories of vitamin E (n = 7; 0.84; 95% CI: 0.71, 0.99) and anthocyanins (n = 2; 0.76; 95% CI: 0.61, 0.96) in cohort studies. Conversely, a significantly higher risk of PD was observed for higher lutein intake (n = 3; 1.86; 95% CI: 1.20, 2.88) among case-control studies.

Dose-response meta-analyses indicated a significant association between a 50-mg/d increase in vitamin C (n = 6; RR: 0.94; 95% CI: 0.88, 0.99), a 5-mg/d increment in vitamin E (n = 7; RR: 0.84; 95% CI: 0.70, 0.99), a 2-mg/d increment in ?-carotene (n = 6; RR: 0.94, 95% CI: 0.89, 0.99), and a 1-mg/d increment in zinc (n = 1; OR: 0.65; 95% CI: 0.49, 0.86) and a reduced risk of PD. Overall, higher intake of antioxidant-rich foods may be associated with a lower risk of PD. 

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery ®
https://www.parkinsonsrecovery.com
rober@parkinsonsrecovery.com

Over the past several months I have received inquiries asking about nicotine as a treatment for Parkinsons. Do I recommend this option out of the over 120 natural options I have determined help reverse Parkinson’s symptoms? Early studies agreed that nicotine is a predictor for a diagnosis of Parkinsons.

The predictive studies use a large data base to ask respondents a number of questions about their dietary and lifestyle habits. Researchers then follow up with respondents after many years – often one or two decades later. These predictive (or sometimes called epidemiological) studies find that smoking does predict whether a person is eventually diagnosed with Parkinsons.

Clinical studies are a very different approach to designing a study. These studies typically select a sample of persons with Parkinsons, then divide the group into an experimental group and a control group. The experimental group uses nicotine patches. The control group gets a patch without the infusion of nicotine.

What do these studies find? The meta analysis below (which is a systematic review of all clinical nicotine studies) does not recommend the use of nicotine patches as a treatment for Parkinsons. Of course smoking is also not recommended

Do I now recommend that you begin using nicotine patches with the anticipation it offers relief from Parkinson’s symptoms? I do know some medical professionals recommend this option.

Given the recent research evidence reported below, I have concluded there are other better options that offer the promise of symptom relief. It is not that the patches will do any harm, but the chances they may help is not promising in light of the recent research evidence.

Recent Studies that examine the impact of Nicotine as a Treatment for Parkinsons

Biomedicines. 2025 Jul 24;13(8):1814. Nicotine Therapy for Parkinson’s Disease: A Meta-Analysis of Randomized Controlled Trials

Abstract

Background: Epidemiological studies have reported an inverse association between smoking and Parkinson’s disease (PD) risk, prompting interest in nicotine as a potential therapeutic agent. The present meta-analysis evaluated the efficacy of nicotine therapy in improving motor symptoms and activities of daily living in patients with PD. 

Methods: PubMed, Embase, and Cochrane Library were systematically searched to identify randomized controlled trials (RCTs) assessing nicotine therapy in PD. Clinical RCTs administering interventions extending beyond 1 week and reporting motor or nonmotor outcomes were included. Random-effects models were used to analyze short-term (<6 months) and long-term (?6 months) outcomes by using standardized mean differences (SMDs). 

Results: This meta-analysis included five RCTs (346 participants). Nicotine therapy led to no significant improvement in motor outcomes in the short term (pooled SMD: -0.452, 95% confidence interval: -1.612 to 0.708) or long term (pooled SMD: 0.174, 95% confidence interval: -0.438 to 0.787). Considerable interstudy heterogeneity was noted. Furthermore, short-term nicotine therapy resulted in no significant improvement in daily functioning, cognition, or quality of life. 

Conclusions: This meta-analysis revealed a lack of compelling evidence suggesting that nicotine-based therapies improve motor or nonmotor outcomes in PD. The findings highlight a disconnect between epidemiological associations and clinical efficacy

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J Parkinsons Dis. 2025 Nov;15(7):1121-1146. Proposed mechanisms of neuroprotection for nicotine in Parkinson’s disease

Abstract

The aim of this review is to summarize the current scientific evidence on nicotine in PD. We describe the large body of scientific articles that address the ways in which nicotine may affect Parkinson’s disease (PD).

Epidemiologic studies have shown that people who smoke have a lower risk of developing PD, and there are multiple potential explanations for why this may be the case. Because nicotine is one active ingredient in tobacco, many studies have used cellular or animal models of PD to try to determine how nicotine may reduce risk of PD or may be of benefit in established PD.

Many of these studies suggest that nicotine can prevent or reduce injury to dopaminergic neurons after exposure to toxic chemicals. However, many questions remain, and clinical trials in which patients with PD are treated with nicotine have largely not shown benefit to patients. Thus, neither smoking nor nicotine treatment are recommended for either prevention or treatment of PD at this time.

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery ®
https://www.parkinsonsrecovery.com
robert@parkinsonsrecovery.com

You are likely thinking – really? Isn’t starch something the laundry puts on my shirts to stiffen them up? The concept of how a resistant starch diet improves Parkinson’s symptoms has a different meaning entirely.

Over the years I have interviewed a variety of health care professionals who recommend one diet or another as a treatment for Parkinsons’s symptoms. The options are overwhelming for most people, given everyone’s body requires different nutrition.

The study below evaluates a resistant starch diet for Parkinsons, indicating surprising success with this approach.

Because everyone’s nutritional requirements are unique to the needs of their body, I recommend you not make any dramatic changes to your diet, especially if it is serving your needs. A few additions may prove beneficial however.

A resistant starch diet involves consuming foods that have dietary fiber. These foods pass through the small intestine largely undigested to nurture gut bacteria. Food sources of a resistant starch diet include unripe fruits (green bananas and green plantains are packed with resistant starch) and vegetables (white beans, kidney beans, black beans, lentils, chickpeas, and green peas).

About a Resistant Starch Diet

Brain Behav Immun. 2026 Feb:132:10621. Resistant starch improves Parkinson’s disease symptoms through restructuring of the gut microbiome and modulating inflammation

Abstract

Alterations in the gut microbiome and a “leaky” gut are associated with Parkinson’s disease (PD), which implies the prospect of rebalancing via dietary intervention. Here, we investigate the impact of a diet rich in resistant starch on the gut microbiome through a multi-omics approach.

We conducted a randomized, controlled trial with short-term and long-term phases involving 74 PD patients of three groups: conventional diet, supplementation with resistant starch, and high-fibre diet.

Our findings reveal associations between dietary patterns and changes in the gut microbiome’s taxonomic composition, functional potential, metabolic activity, and host inflammatory proteome response.

Resistant starch supplementation led to an increase in Faecalibacterium species and short-chain fatty acids alongside a reduction in opportunistic pathogens. Long-term supplementation also increased blood APOA4 and HSPA5 and reduced symptoms of PD. Our study highlights the potential of dietary interventions to modulate the gut microbiome and improve the quality of life for PD patients.

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery
https://www.parkinsonsrecovery.com

Often the simplest solutions are the best. Research below explains how a variety of herbs can be treatments for Parkinsons, in particular rosemary, sage and lavender. The cost is minimal. Potential benefits in offering temporary symptom relief are now well documented.

Specific herbs used to treat symptom of Parkinsons which are covered in the studies posted below were the following (see the full length of the studies for more details):

• Black Cumin
• Cinnamon
• Ginger
• Ginkgo
• Ginseng
• Jiaogulan
• Lavender
• Licorice
• Rosemary
• Safflower
• Saffron
• Sage
• Tumeric
• Mucuna
• Wormwood

These herbs are of course available as supplements in one form or another. You will recognized many as foods available in any health food store.

My suggestion: Begin adding herbs to your meals. They add a boost to the taste and at the same time offer healing properties.

Why Herbs Can Be Treatments for Parkinsons

Nutrients. 2026 Jan 29;18(3):439. Neuroprotective Herbs Associated with Parkinson’s and Alzheimer’s Disease

Abstract

There is currently no treatment for Parkinson’s (PD) and Alzheimer’s (AD) diseases, and medications that target the blockage of amyloid plaque cascades appear to be the most promising for preventing these diseases.

However, it is believed that consuming natural antioxidants, particularly phytochemicals such as phenolic compounds, may help the treatment process for neurodegenerative illnesses. Phenolic substances such as phenolic acids, polyphenols, and flavonoids have been shown to have antioxidant properties in plants and are thought to have a similar impact in humans.

This review provides an analysis of the current landscape of PD and AD pathophysiology, paying particular attention to phytochemical-based therapeutic, preventive, and management strategies using disclosed herb candidates in in vivo/vitro studies. We also highlight the herb-derived components that have recently been identified for their effects in the treatment of PD/AD

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Front Neurosci. 2022 Jun 28:16:909833.  Neuroprotective Potential of Aromatic Herbs: Rosemary, Sage, and Lavender

Abstract

Hundreds of millions of people around the world suffer from neurological disorders or have experienced them intermittently, which has significantly reduced their quality of life. The common treatments for neurological disorders are relatively expensive and may lead to a wide variety of side effects including sleep attacks, gastrointestinal side effects, blood pressure changes, etc.

Several herbal medications have attracted colossal popularity worldwide in the recent years due to their availability, affordable prices, and few side effects. Aromatic plants, sage (Salvia officinalis), lavender (Lavandula angustifolia), and rosemary (Salvia Rosmarinus) have already shown anxiolytics, anti-inflammatory, antioxidant, and neuroprotective effects.

They have also shown potential in treating common neurological disorders, including Alzheimer’s disease, Parkinson’s disease, migraine, and cognitive disorders. This review summarizes the data on the neuroprotective potential of aromatic herbs, sage, lavender, and rosemary.

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery ®
https://www.parkinsonsrecovery.com
robert@parkinsonsrecovery.com

I have a suggestion backed up by the study evidence below that demonstrates how you can improve sleep with a simple solution. No doubt there are dozens of remedies that are proven to support sound sleep to one degree or another.

If you currently do not have a regular exercise routine and have difficulty with getting a good nights sleep, the results in the study posted below suggest that adhering to a strict exercise routine every day has the welcome benefit of improved sleep.

Sleep is critical to recovery. If you seldom drop down into a deep rapid eye movement deep sleep, prospects of recovery are dashed.

If you do have a regular exercise routine and continue to confront sleep challenges, consider increasing the quality and type of exercise you do every day, then see if sleep improves.

Research on Improve Sleep with a Simple Solution

Disabil Rehabil. 2022 Sep;44(19):5585-5591. The association between sleep deficits and sedentary behavior in people with mild Parkinson disease

Abstract

Purpose: Sleep deficits are a common nonmotor symptom of Parkinson disease (PD). People with mild PD also achieve less physical activity (PA) than healthy older adults (HOA), but the relationship between sleep and PA in PD is unclear. This study examined associations between sleep and PA in participants with PD and HOA.

Materials and Methods: Secondary analysis of a prospective observational study. Participants wore a commercially available activity monitor for two weeks. Wilcoxon Rank-Sum tests compared nighttime sleep, wakenings after sleep onset, number of wakenings, naps, step count, and PA intensity between PD and HOA groups. Age-adjusted regression models calculated associations between nighttime sleep and PA.

Results: Per day, participants with PD slept 75 fewer minutes (p < 0.01), took 5,792 fewer steps (p < 0.001), achieved less PA at all intensities, and had 32% more sedentary time (p < 0.001) compared to HOA. Thirty minutes more sleep was associated with 26 fewer sedentary minutes for HOA (p = 0.01) and 25 fewer sedentary minutes for the PD group (p < 0.001).

Conclusions: Sleep and PA are reduced in mild PD compared to HOA. Both groups demonstrated similar associations between reduced sleep and increased sedentary behavior. People with mild Parkinson Disease slept less and were less active than a group of healthy older adults.Less sleep was associated with more sedentary behavior in both groups.The relationship between poor sleep and sedentary behavior in mild Parkinson Disease suggests that rehabilitation interventions may be optimized by targeting both physical activity and sleep deficits.

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery ®
https://www.blog.parkinsonsrecovery.com
robert@parkinsonsrecovery.com

 

After years of intensive research I have documented dozens of natural therapies that offer relief from the symptoms of Parkinson’s disease. When I began my search I expected to find several good therapies at best. The good news is that there are many.

So many in fact, that many members of my audience have become overwhelmed and frustrated with the options. More and more people are asking …

1. Where do I start?
2. Which options are right for me?
3. Which therapies offer the promise of relief from my symptoms?

What has happened is unexpected. Many people do not even know where to start or how. The first step is often never taken due to confusion and frustration over the overload of information about what works and what does not work. Surfing the internet will make your hair fall out! There is just too much information out there for any single person to digest.

I have thoroughly documented these therapies in Road to Recovery from Parkinsons Disease. The good news is that most therapies do offer relief to one extent or another. Some people have better success with some therapies than others. The bad news is that most offer temporary relief.

• What steps can be taken to achieve long lasting relief?
• How can you heal from the inside-out rather than paying lots of money for therapies that offer temporary relief at best?

There is no single therapy, action or habit that does the trick. Many people seek this type of solution and for good reason. We all want to feel better right now. Why wait? Suppressing symptoms can offer temporary relief for some people.

The key can achieving sustained relief from Parkinson’s symptoms is to put the  sympathetic nervous system in the back seat and the parasympathetic nervous system in the driver’s seat. Medicines do not accomplish this, nor do supplements. Simple, easy to do steps must be taken that can offer long lasting relief from symptoms.

This insight has inspired the development of this online course, Giant Leap for Recovery. Is it possible to experience a leap in recovery by embracing a single approach? My answer is no.

One of the reasons more people do not get well is that they are forever looking for that one solution that will fix everything. Such a one stop “fix” to the problem does not exist now nor will it ever exist in the future.

Long lasting relief from symptoms results from taking small steps, one by one. At the end of the journey, you can look back at the starting point and celebrate the progress that has made it possible to take a giant leap in your recovery.

America succeeded in being the first nation to land and walk on the moon by breaking down the problem into tiny parts to be solved. This remarkable feat was not accomplished with a single blast or one amorphous effort. It was accomplished by taking small steps, one by one. Recall the words spoken by Neil Armstrong when he first stepped on the moon:

That’s one small step for man, one giant leap for mankind.

Giant Leap for Recovery Course Details

This online course is offered on a spectacular course platform that presents information for each class in the form of videos, audios, text descriptions and downloads. Everything you need for each class is found in the same place.

I have set up enrollment with a free 7 day trial so you can see learn more about the course. Your credit card is not charged until the 7 day trial ends. The coupon code gift qualifies you for a 50% tuition discount. 

Steps are rolled out every five days. The design invites you to explore and implement recommendations for each of the 14 steps. You can embrace each of the steps yourself. None of them require assistance medical professional or the purchase of therapies.

While you can access the course materials any time of the day or night – you are also invited to always contact me with any follow-up questions or issues that have arisen. I am here to support your journey down the road to recovery. My intention is to offer all the support you need to establish a successful journey down the road to recovery.

Videos, audios, downloads and explanations of the steps are included with each class.

The giant leap is now completed, I want to thank you for this approach. I was surprised by the result, it gave me more hope in finding my own reason of this disease. I am still working on it, after seven years of Parkinson, I only have 2 Sinemet (100/25) per day.

I try to minimize the medication and find a way to heal inside.

Thank you for your support.

All the best for you and your family

Cheers Manon

What are the 14 Steps?

I am not going to tell you, nor explain them in advance. Really you say? Really.

The entire point of Giant Leap for Recovery is to make implementation of each step easy, interesting and fun. I think it best that no one looks ahead at what is coming. After all, taking the challenge of recovery moment to moment is one of the keys to reducing stress! Fixing yourself in the future is one sure fire way to create unnecessary stress. Recovery depends on reducing stress, not inflaming it!

I do want to be clear that all of the steps can be taken with the resources you have available. This has nothing to do with suggesting you purchase and/or take certain supplements or seek out therapists of one type or another. For you to be in the driver’s seat, you need to be in full control of your recovery and not rely on others to fix you.

I can report that you are never going to be able to guess what the 14 steps entail. I am quite sure you have never thought about the usefulness to recovery to many of them. The purpose of each step taken as a whole is to provide the foundation for recovery that can be sustained over the long run.

Click Here to Enroll in Giant Leap for Recovery

Enter the coupon code gift to claim a 50% discount off the regular tuition. Once enrolled, you will have instant access to the course content which kicks off with Step One.

Robert Rodgers PhD
Founder 2004
Parkinsons Recovery ®
Road to Recovery from Parkinsons Disease
https://www.parkinsonsdisease.me
Olympia Washington
877-526-4646