Category: Parkinsons Disease Information(Page 3 of 15)
Access 20 years of research focused on identifying natural approaches, therapies and treatments that offer the opportunity to celebrate relief from the symptoms associated with a diagnosis of Parkinsons disease.
Make 2026 your best year ever by joining my one on one mentoring program. Imagine how differently you will feel after working together with me as we join together to fit together all the pieces of a successful recovery program.
Introducing my One on One Mentorship Program – Your Personal Roadmap to Recovery from neurological challenges. To enroll in the program click on the registration link below.
My mentoring program is all about having the right plan of recovery customized specifically for your unique situation as you celebrate relief from Parkinsons with natural therapies and methods.
What is Included
1. Initial strategy session with me personally to flesh out the potential causes of your symptoms so we can identify therapies and treatments that can successfully address them. This is not some surface-level consultation. It is a deep dive into your specific history and situation. We evaluate what you have tried that did not succeed and what did help. We consider new options.
2. Together we lay out a custom-crafted program of recovery created specifically for your situation.
3. With bimonthly hourly meetings (with a video type interface similar to Zoom or telephone/email contact), we review, update and revise your program of recovery.
We review your progress since our last session
Identify and solve any roadblocks you have confronted.
Refine the recovery program based on real-world results documented by research
Help keep you on track with your program of recovery
Support your progress with new therapeutic and treatment options
4. Unlimited email access to me personally
Why Do I Offer One on One Mentoring
First – After nearly 22 years of researching the causes of Parkinsons and the natural therapies that can reverse them, I have reached a point where my greatest satisfaction in life comes from seeing more and more individuals transform their health.
Second – The individuals I have been mentoring over recent months have been making remarkable progress. This warms my heart and soothes my soul to know that the ongoing attention and support is making a difference.
OK. What is the reason why detoxes flop? Many people with Parkinsons tell me they are frustrated after detoxing for over a year with modest success.
The answer turns on the presence of low sulfur levels that derail the success of any detox program. The body loses its ability to process toxins and medications. Sulfur is needed to synthesize glutathione which is well documented to be seriously deficient in persons diagnosed with Parkinsons.
Glutathione binds to toxins such as heavy metals like mercury and aluminum for extraction from the body. Deficient levels of both sulfur and glutathione increase free radicals and cause cellular damage.
To summarize – sulfur is a critical mineral required for the body’s major detoxification pathways. It plays an essential role in processing and eliminating toxins, medications, hormones, and neurotransmitters. When availability of sulfur is deficient, any detox protocol will be sidelined.
Foods Sources of Sulfur
Dietary sources of sulfur include protein-rich foods like meat, poultry, fish, eggs, and certain vegetables such as garlic and onions. Epsom salt (magnesium sulfate) baths are also a gentle way to absorb sulfate transdermally.
Recent Research that Explains Why Detoxes Flop
Front Nutr. 2025 Apr 9:12:1534000. Natural sulfur compounds in mental health and neurological disorders: insights from observational and intervention studies
Abstract
Over the years, the global disease burden of neurological disorders (NDs) and mental disorders (MDs) has significantly increased, making them one of the most critical concerns and challenges to human health. In pursuit of novel therapies against MD and ND, there has been a growing focus on nutrition and health.
Dietary sulfur, primarily derived from various natural sources, plays a crucial role in numerous physiological processes, including brain function. This review offers an overview of the chemical composition of several natural sources of the sulfur-rich substances such as isothiocyanates, sulforaphane, glutathione, taurine, sulfated polysaccharides, allyl sulfides, and sulfur-containing amino acids, all of which have neuroprotective properties.
A multitude of studies have documented that consuming foods that are high in sulfur enhances brain function by improving cognitive parameters and reduces the severity of neuropathology by exhibiting antioxidant and anti-inflammatory properties at the molecular level.
In addition, the growing role of natural sulfur compounds in repairing endothelial dysfunction, compromising blood-brain barrier and improving cerebral blood flow, are documented here.
This review covers the encouraging results of supplementing sulfur-rich diets in many animal models and clinical investigations, along with their molecular targets in MD, such as schizophrenia, depression, anxiety, bipolar disorder, and autism spectrum disorder, and ND, such as Alzheimer’s disease (AD), Parkinson’s disease (PD), Amyotrophic Lateral Sclerosis (ALS), and Multiple Sclerosis (MS).
The prospects of natural sulfur compounds show great promise as they have potential applications in nutraceuticals, medicines, and functional foods to enhance brain function and prevent diseases.
Many individuals have problems with celebrating restful sleep at night because of tremors that keep them awake. What is the cause of night time tremors which keep people from getting a good night’s sleep?
Many people think the problem of night time tremors is simply a result of a dopamine deficiency. Researchers have recently identified a very different reason for troublesome tremors at night time that keep many people with Parkinsons from getting restful sleeping at night.
This happens to be an issue with the regulation of sugar levels in the blood which is well documented in the research to cause insomnia and sleep deficits.
Low blood sugar levels at night time – a condition called hypoglycemic – contribute to a variety of symptoms including
Tremors
Nervousness
Sweating
Chills
Irritability
Fast heartbeat
Confusion
Headache
Note the overlap of the symptoms of hypoglycemic listed above and those of Parkinsons which include among others tremors, anxiety, irritability, cognitive dysfunction and depression. The synergetic effect of these symptoms result in tremors that keep people awake in the evening or wake them up throughout during the night.
Research Connecting Hypoglycemia with Parkinsons Symptoms
Neurol Neurochir Pol. 2025;59(6):570-575. Episodes of hypoglycemia in patients with Parkinson’s disease – results of two-week continuous glucose monitoring
Abstract
Aim of the study: Recent evidence suggests that Parkinson’s disease (PD) and glucose metabolism disorders may share some common dysregulation pathways. This study sought to determine the incidence of hypoglycemic episodes in PD patients and identify associated risk factors for these events.
Clinical rationale for the study: Hypoglycemia in PD, especially at night, is not well understood. This study quantified hypoglycemia in PD patients and identified autonomic dysfunction as a key risk factor, providing new insights for research into metabolic aspects of PD.
Material and methods: Thirty-five patients diagnosed with PD and 20 control subjects underwent a study consisting of a clinical and biometric assessment and two weeks of continuous glucose monitoring using the Freestyle Libre system to detect episodic hypoglycemia.
Results: Among PD patients, 18 (51.4%) had episodes of hypoglycemia, and 67.8% of the events occurred between 0:00 and 6:00 a.m. Compared to the control group, there were significantly more hypoglycemic episodes registered in PD patients (3.6/pt vs. 1.4/pt; p = 0.0006). Predictors of the occurrence of hypoglycemic episodes in the PD group were lower two-week average glycemic levels (OR: 0.85, p = 0.016), glycemic variability index (OR: 1.46, p = 0.02), and SCOPA-AUT scale score (OR: 1.23, p = 0.04).
Conclusions and clinical implications: Patients with PD are at higher risk of hypoglycemic episodes, especially at night and in the morning. Patients with autonomic dysfunction are at risk for hypoglycemic episodes. Recognizing hypoglycemic episodes can enhance the management of PD by enabling targeted interventions aimed at reducing glycemic variability.
Bacopa monnieri (commonly called bacopa) is a perennial herb that has been used for centuries in Indian Ayurveda traditional medicine. Abstracts of studies included below discuss the many Bacopa benefits for Parkinsons and Alzheimers.
I learned about Bacopa as a treatment for Parkinsons from an interview with herbalist Andrew Bentley. He said …
“There are certain things that are fundamentally helpful with getting the nervous system to repair itself. One of the things I use in that capacity is an herb called Bacopa. Bacopa comes to us from the traditional medicine of India. As such it has a very long documented history of use for helping the nervous system repair itself.”
Recent Studies List A Variety of Bacopa Benefits for Parkinsons.
Nutrients. 2025 Jun 5;17(11):1939. Bacopa monnieri: Preclinical and Clinical Evidence of Neuroactive Effects, Safety of Use and the Search for Improved Bioavailability
Abstract
Bacopa monnieri, also known as Brahmi or Waterhyssop, is a plant used in Ayurveda for its memory-enhancing properties and control of blood sugar levels. It contains active compounds such as alkaloids, saponins, and cucurbitacins, which have various biological activities.
The plant has been studied for its potential in treating Alzheimer’s disease, Parkinson’s disease, attention deficit hyperactivity disorder (ADHD), and depression.
Animal studies have shown promising results in reducing symptoms and protecting against neurodegeneration. Concerning safety, Bacopa monnieri has been found to be generally non-toxic, with no serious side effects reported. However, interactions with certain medications and contraindications in conditions like hyperthyroidism should be considered.
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Curr Pharm Des. 2024;30(13):1016-1030. Investigating Bacopa monnieri L. Therapeutic Potential for the Treatment of Neurological Diseases
Abstract
The popular perennial creeping plant known as Bacopa monnieri (also known as Brahmi) is being utilized in the Indian Ayurvedic medicine practice. It has a variety of bioactive phytoconstituents that have been used therapeutically to treat a number of serious illnesses.
Ancient Vedic scholars used this herb because of its pharmacological effects, particularly as a nerve booster and nootropic supporter. However, it is vital to comprehend the active phytochemical components of Bacopa monnieri extract (BME) and their molecular mechanisms in order to better grasp the effect of BME on neurological illnesses and diseases.
Numerous clinical investigations indicated that BME may have neuroprotective benefits, so it is worthwhile to re-evaluate this wellknown plant.
Here, we focused on neurological problems as we examined the pharmacological and phytochemical characteristics of BME. For their effective usage in neuroprotection and cognition, many clinical concerns and the synergistic potential of Bacopa extract have been investigated.
Alzheimer’s disease is a neurological condition caused by the production of reactive oxygen species, which also causes amyloid-beta (A?) and tau protein aggregation and increases neuro-inflammation and neurotoxicity. Our review offers a more indepth molecular understanding of the neuroprotective functions of BME, which can also be connected to its therapeutic management of neurological illnesses and cognitive-improving effects.
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J Med Food. 2023 Feb;26(2):114-119. Bacopa monnieri in Patients with Parkinson’s Disease: A Pilot Study
Abstract
Purpose of Study: Bacopa monnieri (L.) Wettst. could be of interest in the control of symptoms of Parkinson’s disease, but clinical evidence of its efficacy is lacking. This clinical trial assessed the effects of an extract of B. monnieri on parameters that are related to Parkinson’s diseases.
Design: Twenty volunteers with Parkinson’s disease were recruited for a primary, interventional, controlled, parallel, double-blind clinical study. The volunteers were assigned to treatment with either a commercial B. monnieri extract (225 or 450 mg/day) or placebo. The Parkinson’s Disease Quality-of-Life (PDQL) questionnaire was applied, and motor activity was assessed before treatment and 30, 60, and 90 days after treatment with the B. monnieri extract or placebo. Characteristics such as age, body weight, and height were also collected. No differences in parkinsonian and systemic symptoms, emotional function, or social function were observed between.
Results: The delta percent (?%) showed time-dependent improvements in emotional function with B. monnieri treatment at the daily dose of 450 mg. A strong correlation was found between quality of life and motor outcomes at baseline and 30 days of treatment with B. monnieri, and a moderate correlation for 60 and 90 days of treatment with B. monnieri when compared with baseline time. A moderate correlation was found between motor outcomes and Hoehn and Yahr stages at baseline.
Conclusion. Our results suggest that B. monnieri extract can improve emotional function in Parkinson’s disease patients.
As my video preview below explains, studies report that Cannabidiol CBD offers relief from Parkinson’s symptoms. Annual Parkinsons Recovery surveys also report success with reversing Parkinson’s symptoms when taking CBD oil. It is one of the most frequently listed therapies reported to be useful to members of the Parkinsons Recovery audience.
CBD stands for “cannabidiol oil”. It is extracted from either the Hemp plant or the Marijuana plant. Using CBD allows the medical benefits of medical marijuana without the high that is associated with THC. CBD in itself has no psychedelic properties and is considered safe for consumption
Abstracts of recent CBD studies and posted at the bottom of this post. Taken together, studies find that CBD …
Protects dopaminergic neurons
Reduces neuro-inflammation and oxidative stress
Improves motor symptoms
Increases REM sleep
Reduces insomnia episodes
Enhances mood
Reduces fatigue
Reduces chronic pain
Helpful for stroke victims.
About Alovea Entourage Cannabidiol CBD
Alovea Entourage contain plant-derived cannabinoids, distinct from endocannabinoids that are produced in the body and synthetic cannabinoids. It includes Acemannan, an extract from the Aloe plant, to help the body’s management of mood, memory, sleep and joint health.
SYMBIOTIC IMMUNE SUPPORT
There are many products that have been formulated with additional ingredients to provide support for specific benefits like improved sleep, mood and joint health. One of the most profound impacts has come from the addition of immune modulating aloe vera Acemannan.
Combining the immune modulating fractions of Acemannan with high quality CBD hemp oil provides a symbiotic impact (1 + 1 = 4) that can dramatically enhance overall immune support.
PATENT-PENDING FORMULA INCLUDES:
Organic Broad Spectrum Hemp Oil
Super critical CO2 extraction
Each batch tested to ensure 0.0% THC
Absorption increased by 5-7 times over other hemp oil products with water compatible nanotechnology
Enhanced Entourage Effect through proprietary terpene blend
Symbiotic effect with the highest rated immune modulating Alovéa AE (Acemannan Extract)
Wonderful cinnamon and tropical flavors!
Recent CBD (Cannabidiol) Research Studies
Clinical research on CBD (cannabidiol) includes studies of anxiety, cognition, movement disorders, and pain. Abstracts of recent studies are included below.
Pharmaceuticals (Basel). 2026 Feb 17;19(2):330. Cannabidiol in Neurology: Current Insights and Translational Perspectives
Abstract
Cannabidiol (CBD) is one of the most studied compounds of Cannabis sativa and has attracted significant interest due to its therapeutic and beneficial properties, which have been confirmed in numerous preclinical and clinical studies over the last few years.
A great advantage of CBD over the other widely known Cannabis sativa ingredient, ?-9-tetrahydrocannabinol (THC), is that CBD does not exert intoxicating and psychoactive effects, making it an attractive candidate for therapeutic applications in neurological disorders.
CBD has been shown to exert antioxidant, analgesic, anti-inflammatory, and neuroprotective effects, with therapeutic potential for various neurological conditions. To date, the only drug that consists solely of highly purified CBD is Epidiolex, which is used in the management of severe forms of epilepsy such as Dravet syndrome and Lennox-Gastaut syndrome. Another legal medication containing CBD (albeit with the addition of THC) is Sativex, used to alleviate spasticity in multiple sclerosis.
Besides epilepsy, preclinical data suggest that CBD alone may be potentially beneficial in treating chronic pain, multiple sclerosis, Alzheimer’s and Parkinson’s diseases, or stroke.
The safety profile of CBD is generally considered favorable, as the most commonly reported adverse effects are mild (e.g., somnolence, diarrhea).
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Cannabis Cannabinoid Res. 2026 Feb;11(1):11-29. Unveiling Neurological Benefits: A Review of Hemp Leaf, Flower, Seed Oil Extract, and Their Phytochemical Properties in Neurological Disorders
Abstract
Neurological disorders such as epilepsy, Alzheimer’s disease, Parkinson’s disease, and multiple sclerosis present significant global health care challenges, with complex pathophysiology and limited therapeutic options that often carry substantial side effects.
Hemp-derived compounds, particularly from Cannabis sativa seeds, leaves, and flowers, have gained attention for their potential neuroprotective properties. This review aims to synthesize the current evidence surrounding the therapeutic benefits of hemp-derived compounds, focusing on their bioactive phytochemical profiles, mechanisms of action, and therapeutic efficacy in treating neurological disorders.
A comprehensive review of pre-clinical and clinical studies was conducted, analyzing the phytochemical composition of hemp extracts, including cannabinoids (such as cannabidiol, CBD), terpenes, flavonoids, and polyunsaturated fatty acids. We explored their mechanisms of action through interactions with the endocannabinoid system, neurotransmitter receptors, inflammatory pathways, and oxidative stress mechanisms.
The review highlights the therapeutic potential of hemp-derived extracts in mitigating various neurological conditions. Pre-clinical and clinical studies have demonstrated their efficacy in reducing seizure frequency in epilepsy, protecting dopaminergic neurons in Parkinson’s disease, alleviating neuroinflammation and oxidative stress in Alzheimer’s disease, and promoting remyelination in multiple sclerosis.
The entourage effect, where cannabinoids, terpenes, and flavonoids work synergistically, enhances these therapeutic effects. Innovations in extraction technologies have optimized yield and preserved bioactivity, further enhancing clinical relevance.
Hemp-derived compounds exhibit significant neuroprotective and therapeutic potential for managing neurological disorders.
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J Clin Sleep Med. 2025 Jan 1;21(1):69-80. Effects of a cannabidiol/terpene formulation on sleep in individuals with insomnia: a double-blind, placebo-controlled, randomized, crossover study
Abstract
Study objectives: Cannabidiol (CBD) is increasingly used as a health supplement, though few clinical studies have demonstrated benefits. The primary objective of this study was to evaluate the effects of an oral CBD-terpene formulation on sleep physiology in individuals with insomnia.
Methods: In this double-blind, placebo-controlled, randomized clinical trial, 125 individuals with insomnia received an oral administration of CBD (300 mg) and terpenes (1 mg each of linalool, myrcene, phytol, limonene, ?-terpinene, ?-terpineol, ?-pinene, and ?-caryophyllene) for ? 4 days/wk over 4 weeks using a crossover design. The study medication was devoid of ?9-tetrahydrocannabinol. The primary outcome measure was the percentage of time participants spent in the combination of slow-wave sleep (SWS) and rapid eye movement (REM) sleep stages, as measured by a wrist-worn sleep-tracking device.
Results: This CBD-terpene regimen marginally increased the mean nightly percentage of time participants spent in SWS + REM sleep compared to the placebo (mean [standard error], 1.3% [0.60%]; 95% confidence interval, 0.1-2.5%; P = .03).
More robust increases were observed in participants with low baseline SWS + REM sleep, as well as in day sleepers. For select participants, the increase in SWS + REM sleep averaged as much as 48 minutes/night over a 4-week treatment period. This treatment had no effect on total sleep time, resting heart rate, or heart rate variability, and no adverse events were reported.
Conclusions: Select CBD-terpene ratios may increase SWS + REM sleep in some individuals with insomnia and may have the potential to provide a safe and efficacious alternative to over-the-counter sleep aids and commonly prescribed sleep medications.
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J Obstet Gynaecol Can. 2025 Jul;47(7):102839. Cannabidiol Use and Perceptions of Effectiveness in Women With Chronic Pelvic Pain
Abstract
Objectives: To investigate cannabidiol (CBD) use in women with chronic pelvic pain and nociplastic pain, as represented by a diagnosis of fibromyalgia, including impact on pain and associated symptoms, substitutions for other medications, and side effects.
Methods: This was a cross-sectional study using data derived from an online survey distributed through the National Fibromyalgia Association between April and May of 2020. This secondary analysis was limited to participants with concurrent chronic pelvic pain diagnoses. Participants were asked about their history of CBD use and sub-grouped by use patterns.
Results: Of 1382 included participants, 501 (36.3%) were in the “Never Use” group, 404 (29.2%) were in the “Past Use” but not “Current Use” group and 477 (34.5%) were in the “Current Use” group. Of 477 participants in the “Current Use” group, 80.9% (n = 386) reported pain improvement. The most common indications for current CBD use were pain, insomnia, and anxiety. Participants who reported pain improvement were more likely to report improved sleep (P < 0.001), anxiety (P < 0.001), depression (P = 0.001), fatigue (P < 0.001), overall health (P < 0.001), and substitution of CBD for at least 1 other medication (P = 0.003) than those who reported no pain improvement. About half of the participants reported side effects, most of which were minor with the most common being drowsiness (n = 138, 29%).
Conclusions: Two-thirds of participants with concurrent chronic pelvic pain and fibromyalgia have used CBD. Improved pain with CBD use correlated with improvement in other symptom domains, such as sleep, mood, fatigue, and overall health. A large proportion of those using CBD reported substituting it for higher-risk medications.
Cannabis Cannabinoid Res. 2023 Oct;8(5):716-730.Cannabinoids in Treating Parkinson’s Disease Symptoms: A Systematic Review of Clinical Studies
Abstract
Background: Parkinson’s disease (PD) is a serious neurodegenerative condition impacting many individuals worldwide. There is a need for new non-invasive treatments of PD. Cannabinoids in the form of cannabidiol (CBD) and delta-9-tetrahydrocannabinol (THC) may offer utility as treatment, and our objective was hence to conduct a systematic review regarding the clinical evidence for the efficacy and safety of cannabinoids in treating PD.
Methods: Screening, data extraction, and quality assessments were all conducted by multiple reviewers, with discrepancies resolved by consensus.
Results: After conducting searches in 4 different databases, 673 articles were screened. Thirteen articles were deemed eligible for inclusion in this review. It was shown that cannabis, CBD, and nabilone (a synthetic form of THC) were capable of consistently improving motor symptoms more than a placebo. All treatments improved various non-motor symptoms, particularly with cannabis improving pain intensity, and CBD improving psychiatric symptoms in a dose-dependent manner. Adverse effects were usually minor, and, in the case of CBD, rare (except at very high doses).
Conclusion:Cannabinoids have been shown to safely offer important potential in treating motor symptoms in PD and some non-motor symptoms.
Elderberry, which comes from the Sambucus tree, is a complex of plants native to most of Europe. Common names include elder, elderberry, black elder, European elder, European elderberry, and European black elderberry. Many people are unaware of the elderberry benefits for Parkinsons.
Elderberry (Sambucus nigra) is commonly taken by individuals to support immune function. Studies find it shortens the duration of cold and flu symptoms and reduces their severity. Recent research suggests it is also useful as a treatment for brain health.
Rich in antioxidants like anthocyanins, Elderberry reduces inflammation and fights oxidative stress, both of which are associated with Parkinson’s symptoms.
In light of recent research on Elderberry to treat Parkinson’s symptoms, it is one of the supplements among others that is beneficial to treat Parkinsons symptoms and merits further consideration and discussion with your doctor. This would be especially the case if inflammation is one of the factors identified by you and your doctor that needs to be treated and addressed.
Elderberry is available as an over the counter supplement and does not require a prescription from a doctor.
Research on Elderberry Benefits for Parkinsons
Int J Mol Sci. 2024 Jul 18;25(14):7843. Genus Sambucus (Elderberry): Exploring Its Potential as a Functional Food Ingredient with Neuroprotective Properties Mediated by Antioxidant and Anti-Inflammatory Mechanisms
Abstract
The genus Sambucus, mainly Sambucus nigra, has emerged as a valuable source of bioactive compounds with potential neuroprotective properties. This review explores the antioxidant, anti-inflammatory, and neuroregenerative effects of Sambucus-derived compounds and their implications for brain health and cognitive function.
In vitro studies have demonstrated the ability of Sambucus extracts to mitigate oxidative stress, modulate inflammatory responses, and promote neural stem cell proliferation and differentiation.
In vivo studies using animal models of neurodegenerative diseases, such as Alzheimer’s and Parkinson’s, have shown that Sambucus compounds can improve cognitive function, motor performance, and neuronal survival while attenuating neuroinflammation and oxidative damage.
The neuroprotective effects of Sambucus are primarily attributed to its rich content of polyphenols, particularly anthocyanins, which exert their benefits through multiple mechanisms. These include the modulation of signaling pathways involved in inflammation, apoptosis, mitochondrial function, and oxidative stress.
This review provides a comprehensive overview of the current state of research on the neuroprotective potential of Sambucus and its derivatives, offering valuable insights for the development of dietary strategies to promote brain health and prevent age-related cognitive decline.
Hypomimia or “facial masking” is a restriction in the ability of an individual to express their emotions with facial expressions. The individual has a frozen, blank stare, despite the fact that intense feelings may be experienced. Smile therapy has is shown by research to thaw a frozen face.
Is there any reason to bother with doing smile therapy when facial masking is not an issue for you? My surprise answer is yes. A recent discovery is that smile therapy also improves gait disturbances.
Why in the world might this be the case?
How can flexible movements in a persons face improve gait disturbances?
A primary cause of gait disturbances is a compromised digestive system. As it turns out, the stomach meridian connects multiple point on the face to both feet. We actually do not need to see more evidence evidence to realize improving facial expressions go hand in hand with improving gait disturbances.
What is Smile Therapy?
Smile therapy consists of exercises to facilitate the ability of a person to express their emotions. Exercises encourage the active, voluntary movement of facial muscles to combat facial rigidity.
Smile training involves facial muscle exercises to improve gait speed and reduce slowness of movement. Exercises involve making “surprised” faces and performing “silly” fish-like faces to stretch the muscles around the mouth, eyes and forehead.
Research on the Connection between Smile Therapy and Gait Disturbances
Phys Ther Res. 2024;27(3):173-179. Effects of Smile Training on Gait Disturbance in Parkinson’s Disease Patient with Neuropsychiatric Symptoms: A Single Case Design
Abstract
Objective: To verify the efficacy of smile training in improving gait disturbances in patients with Parkinson’s disease (PD) exhibiting neuropsychiatric symptoms.
Methods: A single-case design with three intervention periods (B1, A1, and B2) was used. During periods B1 and B2, 10 min of smile training (facial muscles training and positive thinking training) was performed before the usual exercise therapy. During the A1 period, the participant received only the usual exercise therapy. During the intervention period, the Timed Up and Go test (TUG) was performed daily in both directions. Tau-U was calculated to determine the effect size of the TUG test time and the number of steps taken during each period.
Movement Disorder Society-Unified Parkinson’s Disease Rating Scale (MDS-UPDRS) Part III, Hospital Anxiety and Depression Scale (HADS), 10-meter walk at maximum speed, Berg Balance Scale, and Characterizing Freezing of Gait Questionnaire (C-FOGQ) were administered on the day before the start of the intervention and the last day of each period.
Results: Comparisons of A1 to B2, TUG time, and the number of steps taken on both turns revealed large reductions (Tau-U ?0.74, p <0.01). The 10-meter walk speed and MDS-UPDRS Part III bradykinesia scores improved, whereas the frequency of gait freezing on the C-FOGQ remained unchanged. The HADS scores did not show significant changes; however, the participant made more positive statements in his reflections.
Conclusion: Smile training may be an effective intervention for improving gait and other motor symptoms in patients with PD.
The mouth is subject to various insults that result in troublesome symptoms such as ulcers, cuts, burns, etc. These can be caused by hot or sharp foods, infections or dental treatments. Suvia mouth wash is a natural treatment with no side effects that protects the health of not only your mouth but your entire digestive system.
Why discuss issues with oral health when the focus of Parkinsons Recovery has always been on natural options to help persons find natural treatments for troublesome neurological challenges?
Answer: The health of the mouth affects the rest of the body. Digestive issues, diabetes, rheumatoid arthritis, cardiovascular diseases and neurodegenerative diseases are well known to be affected by health sores and infections. If you have been unable to bring your digestive system back on line, the problem may well reside with infections in your mouth.
Below is a replay of my interview in 2018 with the developer of Suvia Dr. John Hall who discusses the scientific basis behind the development of Suvia as well as the many uses and benefits provided by this natural way to rejuvenate and regenerate health of the mouth. He makes reference to his formulation as TheraGel. The name has since been changed to Suvia but it is the same mouth wash.
I have personally used Suvia and can report it is the real deal. For more information and to order visit: Suvia
From hot foods to dental care to brushing and flossing to braces and dentures your mouth takes a beating. Suvia adheres to oral tissue, forming a protective barrier that manages pain while maintaining the environment required for optimal wound healing.
Unlike traditional mouthwashes with alcohol and harsh chemicals, Suvia uses 100% natural food ingredients to work with your mouth, not against it.
Suvia provides pain relief, protects wounded areas from contamination, and optimizes healing. It is drug-free and non-toxic and is safe to swallow. Not only does it provide excellent pain relief, but it can be also incorporated into the oral hygiene regimen to help keep the mouth clean and healthy.
Treating anxiety with ashwagandha is a recommendation backed up by sound research evidence. Several recent review studies conclude the herbal remedy ashwagandha reduces anxiety, supports restful sleep and improves overall cognitive functioning.
What is Ashwagandha?
Withania somnifera is an evergreen shrub cultivated in tropical and subtropical areas of Asia, Africa, and Europe. It is called ashwagandha because the plant’s roots smell like a wet horse. The word “ashwa” means horse and the word “gandha” means smell.
Ashwagandha root has been used in the traditional Ayurvedic medicine in India as an adaptogen, a compound that increases a person’s ability to become resilient to biological, physical or chemical stressors. Most of the commercial ashwagandha supplements contain extracts from the plant’s root. Some contain extracts from both the root and leaf.
My Shut Down Anxiety online course covers a variety of different ways to shut down anxiety, the key to celebrating relief from symptoms. Information and course registration is available at:Shut Down Anxiety. Enter the coupon code succeed to claim a 50% discount off of the course tuition.
Studies Recommend Treating Anxiety with Ashwagandha
Results from clinical trials suggest that ashwagandha reduces stress and anxiety. A 2021 systematic review of the research evidence identified seven studies that investigated the use of ashwagandha to treat stress and anxiety.
A total of 491 adults, all from India, with either self-reported high stress and anxiety or a diagnosed anxiety disorder were randomized to take ashwagandha or placebo for 6 to 8 weeks.
Studies found that ashwagandha significantly reduced stress and anxiety levels, reduced fatigue and reduced the stress hormone cortisol when compared with placebo. In several studies, the benefits appeared to be greater with doses of 500 to 600 mg/day than with lower doses.
The more recent meta-analytic reviews of Ashwagandha studies listed below confirm results of the earlier 2021 review summarized above that conclude Ashwagandha has a beneficial impact on shutting down anxiety.
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Explore (NY). 2024 Nov-Dec;20(6):103062. Effects of Ashwagandha (Withania Somnifera) on stress and anxiety: A systematic review and meta-analysis
Abstract
Background: Ashwagandha (Withania somnifera) is an adaptogenic herb used to prevent and treat psychosomatic disorders. This systematic review and meta-analysis evaluated the effects and safety of Ashwagandha on psychosomatic functions related to stress and anxiety among patients.
Methods: A comprehensive search was conducted in MEDLINE, EMBASE, PubMed, PsychINFO, and the Cochrane Library for articles published from January 2000 to January 2022. Randomized controlled trials that examined the effects of Ashwagandha on stress and anxiety were included. Two authors independently extracted all relevant data from the included studies. Both subjective and objective measures of stress and anxiety were assessed as outcome variables.
Result: Nine randomized controlled trials involving 558 patients were eligible for this study. The findings of the meta-analysis showed a significant effect of Ashwagandha formulations on the Perceived Stress Scale (PSS) (MD = -4.72, 95 % CI = [-8.45 to -0.99]), Hamilton Anxiety Scale (HAS) (MD = -2.19, 95 % CI = [-3.83 to -0.55]), and serum cortisol levels (MD = -2.58, 95 % CI = [-4.99 to -0.16]) compared to the placebo group. Among the included studies, four reported mild to moderate adverse events.
Conclusion: The findings from the included studies indicate that Ashwagandha formulations have beneficial effects on stress and anxiety.
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Phytother Res. 2022 Nov;36(11):4115-4124. Does Ashwagandha supplementation have a beneficial effect on the management of anxiety and stress? A systematic review and meta-analysis of randomized controlled trials
Abstract
Background: Clinical trial studies revealed conflicting results on the effect of Ashwagandha extract on anxiety and stress. Therefore, we aimed to evaluate the effect of Ashwagandha supplementation on anxiety as well as stress.
Method: A systematic search was performed in PubMed/Medline, Scopus, and Google Scholar from inception until December 2021. We included randomized clinical trials (RCTs) that investigate the effect of Ashwagandha extract on anxiety and stress. The overall effect size was pooled by random-effects model and the standardized mean difference (SMD) and 95% confidence interval (CIs) for outcomes were applied.
Overall, 12 eligible papers with a total sample size of 1,002 participants and age range between 25 and 48 years were included in the current systematic review and meta-analysis.
Results: We found that Ashwagandha supplementation significantly reduced anxiety (SMD: -1.55, 95% CI: -2.37, -0.74; p = .005; I2 = 93.8%) and stress level (SMD: -1.75; 95% CI: -2.29, -1.22; p = .005; I2 = 83.1%) compared to the placebo. Additionally, the non-linear dose-response analysis indicated a favorable effect of Ashwagandha supplementation on anxiety until 12,000 mg/d and stress at dose of 300-600 mg/d.
Conclusion: The current systematic review and dose-response meta-analysis of RCTs revealed a beneficial effect in both stress and anxiety following Ashwagandha supplementation.
Inflammation is a core feature of Parkinson’s disease, so why not consider natural antioxidants as treatments for Parkinsons? Admittedly, this recommendation may likely be strange to many of you.
Most individuals first diagnosed with Parkinsons focus on dopamine as the primary culprit for their symptoms. This belief leads to the conclusion that the best treatment option is take medicines and supplements that infuse the body with L-dopa, a precursor of dopamine. No other treatment option need be even considered.
Given that studies consistently find inflammation is a core feature of Parkinson’s disease, a companion option is to consider also taking antioxidants as treatments for Parkinsons. Of course, antioxidants can be prescribed by your doctor. A variety of natural antioxidants do not require a prescription.
The following is a list of natural antioxidants that offer the opportunity to celebrate a relief from symptoms inflamed by inflammation:
N-acetylcysteine
sulforaphane
alpha-lipoic acid
L-carnitine
ascorbic acid (Vitamin C)
selenocompounds
flavones
zinc
Vitamin E
Why not select a few of the above natural antioxidants to take on a regular basis to keep inflammation from overcoming the body’s ability to rejuvenate and regenerate?
Research on Antioxidants as Treatments for Parkinsons
Front Neurosci. 2024 Dec 5:18:1505153. Antioxidants in neuropsychiatric disorder prevention: neuroprotection, synaptic regulation, microglia modulation, and neurotrophic effect
Abstract
Oxidative stress, caused by an imbalance between the generation of reactive oxygen species (ROS) and the body’s intrinsic antioxidant defenses, plays a critical role in neurodegenerative diseases such as Alzheimer’s, Parkinson’s, and Huntington’s. Beyond these conditions, recent evidence indicates that dysregulated redox balance is implicated in neuropsychiatric disorders, including schizophrenia, major depressive disorder, and anxiety disorders.
Preclinical and clinical studies have demonstrated the potential of antioxidants, such as N-acetylcysteine, sulforaphane, alpha-lipoic acid, L-carnitine, ascorbic acid, selenocompounds, flavones and zinc, in alleviating neuropsychiatric symptoms by mitigating excitotoxicity, enhancing synaptic plasticity, reducing microglial overactivation and promoting synaptogenesis.
This review explores the role of oxidative stress in the pathogenesis of neuropsychiatric disorders. It provides an overview of the current evidence on antioxidant therapy’s pharmacological effects, as demonstrated in animal models and clinical studies.
Given the limitations and side effects of existing treatments for neuropsychiatric disorders, antioxidant therapy presents a promising, safer alternative. Further research is essential to deepen our understanding and investigate the clinical efficacy and mechanisms underlying these therapies.
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Adv Nutr. 2022 Oct 2;13(5):1493-1504. Dietary Antioxidants and Risk of Parkinson’s Disease: A Systematic Review and Dose-Response Meta-analysis of Observational Studies
Abstract
The aim of the current review was to explore the association between various dietary antioxidants and the risk of developing Parkinson’s disease (PD).
PubMed, Scopus, Web of Science, and Google Scholar were searched up to March 2021. Prospective, observational cohort studies, nested case-control, and case-control designs that investigated the association between antioxidants and PD risk were included. A random-effects model was used to pool the RRs. The certainty of the evidence was rated using the GRADE (Grading of Recommendations Assessment, Development, and Evaluations) scoring system. In addition, a dose-response relation was examined between antioxidant intake and PD risk.
Six prospective cohort studies and 2 nested case-control (total n = 448,737 with 4654 cases), as well as 6 case-control (1948 controls, 1273 cases) studies were eligible. The pooled RR was significantly lower for the highest compared with the lowest intake categories of vitamin E (n = 7; 0.84; 95% CI: 0.71, 0.99) and anthocyanins (n = 2; 0.76; 95% CI: 0.61, 0.96) in cohort studies. Conversely, a significantly higher risk of PD was observed for higher lutein intake (n = 3; 1.86; 95% CI: 1.20, 2.88) among case-control studies.
Dose-response meta-analyses indicated a significant association between a 50-mg/d increase in vitamin C (n = 6; RR: 0.94; 95% CI: 0.88, 0.99), a 5-mg/d increment in vitamin E (n = 7; RR: 0.84; 95% CI: 0.70, 0.99), a 2-mg/d increment in ?-carotene (n = 6; RR: 0.94, 95% CI: 0.89, 0.99), and a 1-mg/d increment in zinc (n = 1; OR: 0.65; 95% CI: 0.49, 0.86) and a reduced risk of PD. Overall, higher intake of antioxidant-rich foods may be associated with a lower risk of PD.
Over the past several months I have received inquiries asking about nicotine as a treatment for Parkinsons. Do I recommend this option out of the over 120 natural options I have determined help reverse Parkinson’s symptoms? Early studies agreed that nicotine is a predictor for a diagnosis of Parkinsons.
The predictive studies use a large data base to ask respondents a number of questions about their dietary and lifestyle habits. Researchers then follow up with respondents after many years – often one or two decades later. These predictive (or sometimes called epidemiological) studies find that smoking does predict whether a person is eventually diagnosed with Parkinsons.
Clinical studies are a very different approach to designing a study. These studies typically select a sample of persons with Parkinsons, then divide the group into an experimental group and a control group. The experimental group uses nicotine patches. The control group gets a patch without the infusion of nicotine.
What do these studies find? The meta analysis below (which is a systematic review of all clinical nicotine studies) does not recommend the use of nicotine patches as a treatment for Parkinsons. Of course smoking is also not recommended
Do I now recommend that you begin using nicotine patches with the anticipation it offers relief from Parkinson’s symptoms? I do know some medical professionals recommend this option.
Given the recent research evidence reported below, I have concluded there are other better options that offer the promise of symptom relief. It is not that the patches will do any harm, but the chances they may help is not promising in light of the recent research evidence.
Recent Studies that examine the impact of Nicotine as a Treatment for Parkinsons
Biomedicines. 2025 Jul 24;13(8):1814. Nicotine Therapy for Parkinson’s Disease: A Meta-Analysis of Randomized Controlled Trials
Abstract
Background: Epidemiological studies have reported an inverse association between smoking and Parkinson’s disease (PD) risk, prompting interest in nicotine as a potential therapeutic agent. The present meta-analysis evaluated the efficacy of nicotine therapy in improving motor symptoms and activities of daily living in patients with PD.
Methods: PubMed, Embase, and Cochrane Library were systematically searched to identify randomized controlled trials (RCTs) assessing nicotine therapy in PD. Clinical RCTs administering interventions extending beyond 1 week and reporting motor or nonmotor outcomes were included. Random-effects models were used to analyze short-term (<6 months) and long-term (?6 months) outcomes by using standardized mean differences (SMDs).
Results: This meta-analysis included five RCTs (346 participants). Nicotine therapy led to no significant improvement in motor outcomes in the short term (pooled SMD: -0.452, 95% confidence interval: -1.612 to 0.708) or long term (pooled SMD: 0.174, 95% confidence interval: -0.438 to 0.787). Considerable interstudy heterogeneity was noted. Furthermore, short-term nicotine therapy resulted in no significant improvement in daily functioning, cognition, or quality of life.
Conclusions:This meta-analysis revealed a lack of compelling evidence suggesting that nicotine-based therapies improve motor or nonmotor outcomes in PD. The findings highlight a disconnect between epidemiological associations and clinical efficacy
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J Parkinsons Dis. 2025 Nov;15(7):1121-1146. Proposed mechanisms of neuroprotection for nicotine in Parkinson’s disease
Abstract
The aim of this review is to summarize the current scientific evidence on nicotine in PD. We describe the large body of scientific articles that address the ways in which nicotine may affect Parkinson’s disease (PD).
Epidemiologic studies have shown that people who smoke have a lower risk of developing PD, and there are multiple potential explanations for why this may be the case. Because nicotine is one active ingredient in tobacco, many studies have used cellular or animal models of PD to try to determine how nicotine may reduce risk of PD or may be of benefit in established PD.
Many of these studies suggest that nicotine can prevent or reduce injury to dopaminergic neurons after exposure to toxic chemicals. However, many questions remain, and clinical trials in which patients with PD are treated with nicotine have largely not shown benefit to patients. Thus, neither smoking nor nicotine treatment are recommended for either prevention or treatment of PD at this time.
You are likely thinking – really? Isn’t starch something the laundry puts on my shirts to stiffen them up? The concept of how a resistant starch diet improves Parkinson’s symptoms has a different meaning entirely.
Over the years I have interviewed a variety of health care professionals who recommend one diet or another as a treatment for Parkinsons’s symptoms. The options are overwhelming for most people, given everyone’s body requires different nutrition.
The study below evaluates a resistant starch diet for Parkinsons, indicating surprising success with this approach.
Because everyone’s nutritional requirements are unique to the needs of their body, I recommend you not make any dramatic changes to your diet, especially if it is serving your needs. A few additions may prove beneficial however.
Review the list of foods below. Why not consider adding one or several to your meals? The study posted below suggests making a few additions to your regular diet may well prove beneficial in offering relief from Parkinson’s symptoms.
A resistant starch diet involves consuming foods that have dietary fiber. These foods pass through the small intestine largely undigested to nurture gut bacteria. Food sources of a resistant starch diet include unripe fruits (green bananas and green plantains are packed with resistant starch) and vegetables (white beans, kidney beans, black beans, lentils, chickpeas, and green peas).
About a Resistant Starch Diet
Brain Behav Immun. 2026 Feb:132:10621. Resistant starch improves Parkinson’s disease symptoms through restructuring of the gut microbiome and modulating inflammation
Abstract
Alterations in the gut microbiome and a “leaky” gut are associated with Parkinson’s disease (PD), which implies the prospect of rebalancing via dietary intervention. Here, we investigate the impact of a diet rich in resistant starch on the gut microbiome through a multi-omics approach.
We conducted a randomized, controlled trial with short-term and long-term phases involving 74 PD patients of three groups: conventional diet, supplementation with resistant starch, and high-fibre diet.
Our findings reveal associations between dietary patterns and changes in the gut microbiome’s taxonomic composition, functional potential, metabolic activity, and host inflammatory proteome response.
Resistant starch supplementation led to an increase in Faecalibacterium species and short-chain fatty acids alongside a reduction in opportunistic pathogens. Long-term supplementation also increased blood APOA4 and HSPA5 and reduced symptoms of PD. Our study highlights the potential of dietary interventions to modulate the gut microbiome and improve the quality of life for PD patients.
Often the simplest solutions are the best. Research below explains how a variety of herbs can be treatments for Parkinsons, in particular rosemary, sage and lavender. The cost is minimal. Potential benefits in offering temporary symptom relief are now well documented.
Specific herbs used to treat symptom of Parkinsons which are covered in the studies posted below were the following (see the full length of the studies for more details):
Black Cumin
Cinnamon
Ginger
Ginkgo
Ginseng
Jiaogulan
Lavender
Licorice
Rosemary
Safflower
Saffron
Sage
Tumeric
Mucuna
Wormwood
These herbs are of course available as supplements in one form or another. You will recognized many as foods available in any health food store.
My suggestion: Begin adding herbs to your meals. They add a boost to the taste and at the same time offer healing properties.
Why Herbs Can Be Treatments for Parkinsons
Nutrients. 2026 Jan 29;18(3):439. Neuroprotective Herbs Associated with Parkinson’s and Alzheimer’s Disease
Abstract
There is currently no treatment for Parkinson’s (PD) and Alzheimer’s (AD) diseases, and medications that target the blockage of amyloid plaque cascades appear to be the most promising for preventing these diseases.
However, it is believed that consuming natural antioxidants, particularly phytochemicals such as phenolic compounds, may help the treatment process for neurodegenerative illnesses. Phenolic substances such as phenolic acids, polyphenols, and flavonoids have been shown to have antioxidant properties in plants and are thought to have a similar impact in humans.
This review provides an analysis of the current landscape of PD and AD pathophysiology, paying particular attention to phytochemical-based therapeutic, preventive, and management strategies using disclosed herb candidates in in vivo/vitro studies. We also highlight the herb-derived components that have recently been identified for their effects in the treatment of PD/AD
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Front Neurosci. 2022 Jun 28:16:909833. Neuroprotective Potential of Aromatic Herbs: Rosemary, Sage, and Lavender
Abstract
Hundreds of millions of people around the world suffer from neurological disorders or have experienced them intermittently, which has significantly reduced their quality of life. The common treatments for neurological disorders are relatively expensive and may lead to a wide variety of side effects including sleep attacks, gastrointestinal side effects, blood pressure changes, etc.
Several herbal medications have attracted colossal popularity worldwide in the recent years due to their availability, affordable prices, and few side effects. Aromatic plants, sage (Salvia officinalis), lavender (Lavandula angustifolia), and rosemary (Salvia Rosmarinus) have already shown anxiolytics, anti-inflammatory, antioxidant, and neuroprotective effects.
They have also shown potential in treating common neurological disorders, including Alzheimer’s disease, Parkinson’s disease, migraine, and cognitive disorders. This review summarizes the data on the neuroprotective potential of aromatic herbs, sage, lavender, and rosemary.
Robert Rodgers PhD Founder 2004 Parkinsons Recovery ® https://www.parkinsonsrecovery.com robert@parkinsonsrecovery.com
I have a suggestion backed up by the study evidence below that demonstrates how you can improve sleep with a simple solution. No doubt there are dozens of remedies that are proven to support sound sleep to one degree or another.
If you currently do not have a regular exercise routine and have difficulty with getting a good nights sleep, the results in the study posted below suggest that adhering to a strict exercise routine every day has the welcome benefit of improved sleep.
Sleep is critical to recovery. If you seldom drop down into a deep rapid eye movement deep sleep, prospects of recovery are dashed.
If you do have a regular exercise routine and continue to confront sleep challenges, consider increasing the quality and type of exercise you do every day, then see if sleep improves.
Research on Improve Sleep with a Simple Solution
Disabil Rehabil. 2022 Sep;44(19):5585-5591. The association between sleep deficits and sedentary behavior in people with mild Parkinson disease
Abstract
Purpose: Sleep deficits are a common nonmotor symptom of Parkinson disease (PD). People with mild PD also achieve less physical activity (PA) than healthy older adults (HOA), but the relationship between sleep and PA in PD is unclear. This study examined associations between sleep and PA in participants with PD and HOA.
Materials and Methods: Secondary analysis of a prospective observational study. Participants wore a commercially available activity monitor for two weeks. Wilcoxon Rank-Sum tests compared nighttime sleep, wakenings after sleep onset, number of wakenings, naps, step count, and PA intensity between PD and HOA groups. Age-adjusted regression models calculated associations between nighttime sleep and PA.
Results: Per day, participants with PD slept 75 fewer minutes (p < 0.01), took 5,792 fewer steps (p < 0.001), achieved less PA at all intensities, and had 32% more sedentary time (p < 0.001) compared to HOA. Thirty minutes more sleep was associated with 26 fewer sedentary minutes for HOA (p = 0.01) and 25 fewer sedentary minutes for the PD group (p < 0.001).
Conclusions: Sleep and PA are reduced in mild PD compared to HOA. Both groups demonstrated similar associations between reduced sleep and increased sedentary behavior. People with mild Parkinson Disease slept less and were less active than a group of healthy older adults.Less sleep was associated with more sedentary behavior in both groups.The relationship between poor sleep and sedentary behavior in mild Parkinson Disease suggests that rehabilitation interventions may be optimized by targeting both physical activity and sleep deficits.
Click on the image to the left to enroll in my three month Giant Leap for Recovery Online Course.
Enter the coupon code gift to claim a 50% tuition discount.
After years of intensive research I have documented dozens of natural therapies that offer relief from the symptoms of Parkinson’s disease. When I began my search I expected to find several good therapies at best. The good news is that there are many.
So many in fact, that many members of my audience have become overwhelmed and frustrated with the options. More and more people are asking …
Where do I start?
Which options are right for me?
Which therapies offer the promise of relief from my symptoms?
What has happened is unexpected. Many people do not even know where to start or how. The first step is often never taken due to confusion and frustration over the overload of information about what works and what does not work. Surfing the internet will make your hair fall out! There is just too much information out there for any single person to digest.
I have thoroughly documented these therapies in Road to Recovery from Parkinsons Disease. The good news is that most therapies do offer relief to one extent or another. Some people have better success with some therapies than others. The bad news is that most offer temporary relief.
What steps can be taken to achieve long lasting relief?
How can you heal from the inside-out rather than paying lots of money for therapies that offer temporary relief at best?
There is no single therapy, action or habit that does the trick. Many people seek this type of solution and for good reason. We all want to feel better right now. Why wait? Suppressing symptoms can offer temporary relief for some people.
The key can achieving sustained relief from Parkinson’s symptoms is to put the sympathetic nervous system in the back seat and the parasympathetic nervous system in the driver’s seat. Medicines do not accomplish this, nor do supplements. Simple, easy to do steps must be taken that can offer long lasting relief from symptoms.
This insight has inspired the development of this online course, Giant Leap for Recovery. Is it possible to experience a leap in recovery by embracing a single approach? My answer is no.
One of the reasons more people do not get well is that they are forever looking for that one solution that will fix everything. Such a one stop “fix” to the problem does not exist now nor will it ever exist in the future.
Long lasting relief from symptoms results from taking small steps, one by one. At the end of the journey, you can look back at the starting point and celebrate the progress that has made it possible to take a giant leap in your recovery.
America succeeded in being the first nation to land and walk on the moon by breaking down the problem into tiny parts to be solved. This remarkable feat was not accomplished with a single blast or one amorphous effort. It was accomplished by taking small steps, one by one. Recall the words spoken by Neil Armstrong when he first stepped on the moon:
That’s one small step for man, one giant leap for mankind.
Giant Leap for Recovery Course Details
This online course is offered on a spectacular course platform that presents information for each class in the form of videos, audios, text descriptions and downloads. Everything you need for each class is found in the same place.
I have set up enrollment with a free 7 day trial so you can see learn more about the course. Your credit card is not charged until the 7 day trial ends. The coupon code gift qualifies you for a 50% tuition discount.
Steps are rolled out every five days. The design invites you to explore and implement recommendations for each of the 14 steps. You can embrace each of the steps yourself. None of them require assistance medical professional or the purchase of therapies.
While you can access the course materials any time of the day or night – you are also invited to always contact me with any follow-up questions or issues that have arisen. I am here to support your journey down the road to recovery. My intention is to offer all the support you need to establish a successful journey down the road to recovery.
Videos, audios, downloads and explanations of the steps are included with each class.
The giant leap is now completed, I want to thank you for this approach. I was surprised by the result, it gave me more hope in finding my own reason of this disease. I am still working on it, after seven years of Parkinson, I only have 2 Sinemet (100/25) per day.
I try to minimize the medication and find a way to heal inside.
Thank you for your support.
All the best for you and your family
Cheers Manon
What are the 14 Steps?
I am not going to tell you, nor explain them in advance. Really you say? Really.
The entire point of Giant Leap for Recoveryis to make implementation of each step easy, interesting and fun. I think it best that no one looks ahead at what is coming. After all, taking the challenge of recovery moment to moment is one of the keys to reducing stress! Fixing yourself in the future is one sure fire way to create unnecessary stress. Recovery depends on reducing stress, not inflaming it!
I do want to be clear that all of the steps can be taken with the resources you have available. This has nothing to do with suggesting you purchase and/or take certain supplements or seek out therapists of one type or another. For you to be in the driver’s seat, you need to be in full control of your recovery and not rely on others to fix you.
I can report that you are never going to be able to guess what the 14 steps entail. I am quite sure you have never thought about the usefulness to recovery to many of them. The purpose of each step taken as a whole is to provide the foundation for recovery that can be sustained over the long run.
Enter the coupon code giftto claim a 50% discount off the regular tuition. Once enrolled, you will have instant access to the course content which kicks off with Step One.
Robert Rodgers PhD Founder 2004 Parkinsons Recovery ® Road to Recovery from Parkinsons Disease https://www.parkinsonsdisease.me Olympia Washington 877-526-4646
The question “can you reverse Parkinson’s disease” is answered for you here.
What really matters when it comes down to a successful program of recovery?
What needs to happen to celebrate a reversal of Parkinson’s disease?
Results of my annual Parkinsons Recovery survey answer the question posed here – can you reverse Parkinson’s disease. What survey you ask?
Parkinsons Recovery Annual Survey
Each year during the holiday season I send out a question survey to members of the global Parkinsons Recovery audience. The question asked on the survey was:
Please list below natural therapies that have helped you experience relief from symptoms of Parkinson’s disease
I am always surprised and awed with the variety and diversity of therapies reported to have been helpful by members of the Parkinsons Recovery audience. When I examined results of the survey, I asked myself the question – what is the common theme here?
The answer quickly became crystal clear. Therapies reported to have been useful in reversing Parkinson’s symptoms turn down the volume of the sympathetic nervous system (which inflames symptoms) and switch on the parasympathetic (which soothes symptoms).
There is no single approach, method or therapy that permanently flips the switch. Each person discovers their own unique way to make this happen for themselves.
Dozens of therapies are reported to have been useful in this regard as reflected in my survey. They are now thoroughly listed and documented in my online course What Really Matters that rolls out a comprehensive explanation and review of each recovery option. Click on the What Really Matters Link below to register and take advantage of the 7 day free trial.
While there are a surprising number of natural therapies that help, I wanted you to know about several in particular. Which ones among the many natural therapies will offer relief from your Parkinson’s symptoms? Consider the following possibilities that are reviewed in detail in this course.
Tumeric
Turmeric helps prevent depletion of dopamine, reduces oxidative stress and reverses inflammation
Magnesium Magnesium is a natural muscle relaxant which helps address issues with rigidity and constipation.
Lion’s Mane Mushrooms Lion’s Mane Mushrooms show welcome benefits as a therapy to reverse symptoms
Pulsed Electromagnetic Field Therapy (PEMF) One reason detoxes are unsuccessful is compromised circulation. PEMF is an ideal therapy to address this problem.
Passion Flower Some persons who experience neurological symptoms report Passion Flower has been helpful
Online Course Information
The new What Really Matters online course rolls out a comprehensive explanation and review of each recovery option. Click on the What Really Matters Link below to register and take advantage of the 7 day free trial.
Fasting Fasting is a natural way for the body to produce ketones which are the most efficient fuel for our brains.
Eliminate Dairy Research consistently reports that eating dairy is a significant predictor of who experiences symptoms of Parkinson’s.
CBD Oil Research studies report that CBD helps to shut down anxiety. Once anxiety is brought under control, relief of symptoms follows.
Light Therapy – Photobiomodulation Reports from persons who experience symptoms of Parkinson’s indicate light therapy has a beneficial impact on symptoms for 9 out of 10 users
Fava Beans
Aunt Bean has formulated a home made fava bean tincture that offers welcome relief from her Parkinson’s symptoms. Here is the recipe so you can prepare it for yourself.
Food as Medicine Do you insist on only eating organic food? Great, but guess what? Eating organic is not enough. The food necessary for cellular healing is actually not available or sold in most food outlets.
N-Acetylcysteine (NAC) N-Acetylcysteine (NAC) is a natural anti-oxidant that boosts glutathione levels which are usually dangerously low in persons diagnosed with Parkinson’s disease.
Low Dose Naltrezone (LDN) Low Dose Naltrezone (LDN) is a prescription medicine that boosts the immune system. It is reported by some persons diagnosed with Parkinson’s to have been beneficial.
Hot Ginger Spine Therapy Macrobiotic Guru Warren Kramer’s Hot Ginger Spine Therapy
Secret to Recovery When most people get sick they immediately gravitate toward finding something they can put into their body, but …
Importance of Balance in Diet and Lifestyle Excesses in diet and lifestyle sustain tremors. When you eat mostly meat or carbs – you are out of balance. When you work 24/7 you are out of balance.
Inclined Bed Therapy Improve circulation while you sleep by raising the head of your bed 6-8 inches.
Infusion of Energy An Exercise to Kick Start Your Energy using your siren voice along with a simple movement of arms and body.
Fermented Papaya
Three Types of Toxins There is no such thing as a single detox protocol that gets rid of any and all toxins. So, which therapies should be used for which toxins?
Heavy Metal Toxicity Types of heavy metal toxicity and methods to detox them
Problems with Tying Shoe Laces or Buttoning a Shirt? Devices designed to make typing shoe laces or buttoning your shirt easier may not be in your best and highest good.
Yoga Many individuals who experience symptoms of Parkinson’s report Yoga has been a life saver.
Emotion Code
Dozens of natural options have been shown to offer relief from symptoms of Parkinson’s disease. The listing of possibilities above offers a sneak preview of the many shown to be helpful.
Summary: Can You Reverse Parkinson’s Disease
My answer to the question is obviously yes.
Dozens options like the ones listed above are rolled out in my new “What Really Matters” online course. Each option is thoroughly documented, accompanied by the research evidence that evaluates each option’s efficacy for persons who experience Parkinson’s symptoms. Selections are inspired by what members of my audience report has helped them get relief from their symptoms. I list each option in the course because I know it has helped some people reverse their symptoms.
Click on the What Really Matters Link below to register and take advantage of the 7 day free trial.
In this video I preview a marvelous suggestion for how to manifest rock solid walking.
Here is the clip of my interview with John Pepper.
This suggestion to manifest centered and balanced walking is one among – get this – 144 other therapies, techniques and approaches to manifest rock solid walking covered in my Rock Solid Walking online course. The ideas and techniques are drawn from 275 interviews I have hosted over the past two decades. Visit the link below for more information about the course:
John Pepper, author of Reverse Parkinsons Disease, is a Parkinson’s Patient with six decades of experience. What has helped John manifest recovery is:
Taking Conscious Control of all movements
Doing Energetic Exercise
Managing Stress
Adopting a Positive Attitude
Below is the study which reports on the benefits of conscious control of movements to manifest Rock Solid Walking.
Neurorehabil Neural Repair. 2007 Mar-Apr;21(2):123-6. Duration of Parkinson disease is associated with an increased propensity for “reinvestment”. R S W Masters, H S Pall, K M A MacMahon, F F Eves
Abstract
Background: As a consequence of difficulties in movement initiation and execution, people with Parkinson’s disease (PD) are typically encouraged to consciously monitor and control the mechanics of their actions. This is described as ‘reinvestment’ and has been shown to help mediate effective motor output.
Objective: To examine the propensity for reinvestment in a sample of people with PD.
Methods: A volunteer sample of 55 people with PD was asked to complete a previously validated measure, the Reinvestment Scale. A sub-sample (and age matched controls) was asked to complete a recently developed, movement specific, version of the Scale. Data was collected on Mini Mental State Examination and the Hoehn & Yahr Scale. Participant demographics, including age of onset and duration of disease, were also collated.
Results: Multiple regression analyses showed that duration of disease was associated with reinvestment score on both the Reinvestment Scale and the Movement Specific Reinvestment Scale.
Conclusions: Participants appeared to become more aware of the mechanics of their actions over time. Possible explanations for this finding are discussed with reference to rehabilitation.
Studies have connected symptoms of Parkinsons to a surprising variety of infections. Included in this list is a candida yeast infection.
Testimonials from members of my audience lead me to conclude a yeast infection presents formidable challenges to control. A little yeast is beneficial for digestion. Too much wreaks havoc on a neurological system.
Candida treatments potentially offer ways to control an overgrowth in candida. The most successful long term approach however is to avoid eating sugar of any form. Why?
Sugar is the food that the candida yeast infection thrives on. It sets the conditions for the existing overgrowth to multiply many times over. Overgrowth can be a temporary inconvenience or, if fed with sugar, transform into a chronic set of neurological symptoms.
Below is my interview with Lydia Epp who to my knowledge is the first researcher to identify the connection between Candida and Parkinsons.
Candida is a yeast overgrowth in the intestines and mucous membranes and leaks into the bloodstream. It normally exists in small amounts in the body, particularly in areas like the mouth, gut, and vagina. However, an overgrowth can lead to infections, known as candidiasis.
This can manifest in various forms, such as oral thrush, vaginal yeast infections, and systemic infections in immunocompromised individuals. Factors like antibiotic use, a weakened immune system, and diabetes can contribute to its overgrowth.
It is a live fungus caused by long term use of antibiotics, steroids, oral contraceptives and most predominately a diet rich in carbs and sugars.
Problems result with candida when it colonizes into a critical mass as it replaces the good bacteria and creates havoc with the digestive system.
Candida Testimonial
I have also made the connection of candida and Parkinsons disease. I was on tetracycline for acne as a young teen and suffered with yeast infections off and on that exploded into severe, chronic infections after my dad died. Diflucan for over 18 months couldn’t stop; foot soaks held the infections at bay and within 2 years I was dx with PD.
I take no meds and have spent 8 years on mind-body-emotions work, healing techniques, attended your Santa Fe seminar… I’m 6 days into a strict no sugar, no gluten diet and feel more energetic and my ankles are skinny again.
Dance with Candida
The dance with candida never ends. In the end it is a question of balance. Too much or too little candida results in horrible health problems. some of which include the symptoms of Parkinsons.
I have done many different candida detoxes over the years. They all have helped to reduce the infection in the tissues and organs of my body. It does pay to stay in a strict no sugar candida diet for 6 weeks every now and then. The problem is never really permanently solved as long as a diet is dominated by carbs and sugars.
I instantly know candida is an issue for me personally because I get fatigued easily. I also know I have a candida overgrowth because I have indulged in consuming sweets. I also know because rashes can also pop up here and there on my body. There is no need to request diagnostic tests for a candida problem. My body always sends off the blinking red light alarm.
In the long run healing an addiction to sugar is the key to getting the candida problem under control.
Connection of Candida to Parkinsons
Most people think that all toxins enter the body externally through the food we eat and through toxins that enter our body through our skin. Some toxins are generated internally by the body. Candida is one substance that is responsible directly or indirectly for the production of 50 toxins.
One such toxin is acetaldehyde which is typically found in the gastro-intestional track. Another toxin – salsolinol – is synthesized by the body from acedaldehyde and dopamine. Elevated levels of salsolinol have been found in the urine and cerebrospinal fluid of persons who experience Parkinson’s symptoms.
Will you be likely to remember these complicated terms and connections a week from now or even tomorrow? I doubt it. What is important to remember is that evidence suggests an overgrowth of candida can be an agent responsible for Parkinson’s symptoms.
Lidia Epp is the researcher who discovered the connection between Candida and Parkinson’s symptoms. She is one of the contributors to Pioneers of Recovery. Her Parkinson’s symptoms reversed after completing a successful candida cleanse.
Learning how to dance successfully with candida is the same lesson as learning to balance the energies of giving and receiving. Many people with Parkinson’s symptoms are givers who never stop to receive. They give their life force away. Learning to maintain a delicate balance of giving to others and receiving from others is one of the secrets to a successful recovery program. It is no less important than learning to balance the level of candida in our body.
Below are Abstracts of Two Studies that Connect Candida to Parkinsons
Int Biol Sci. 2020 Feb 10;16(7):1135-1152. Parkinson’s Disease: A Comprehensive Analysis of Fungi and Bacteria in Brain Tissue
Abstract
Parkinson’s disease (PD) is characterized by motor disorders and the destruction of dopaminergic neurons in the substantia nigra pars compacta. In addition to motor disability, many patients with PD present a spectrum of clinical symptoms, including cognitive decline, psychiatric alterations, loss of smell and bladder dysfunction, among others. Neuroinflammation is one of the most salient features of PD, but the nature of the trigger remains unknown.
A plausible mechanism to explain inflammation and the range of clinical symptoms in these patients is the presence of systemic microbial infection. Accordingly, the present study provides extensive evidence for the existence of mixed microbial infections in the central nervous system (CNS) of patients with PD.
Assessment of CNS sections by immuno-histochemistry using specific antibodies revealed the presence of both fungi and bacteria. Moreover, different regions of the CNS were positive for a variety of microbial morphologies, suggesting infection by a number of microorganisms.
Identification of specific fungal and bacterial species in different CNS regions from six PD patients was accomplished using nested PCR analysis and next-generation sequencing, providing compelling evidence of polymicrobial infections in the CNS of PD. Most of the fungal species identified belong to the general were Streptococcus and Pseudomonas, with most bacterial species belonging to the phyla Actinobacteria and ProteobacteriaBotrytis, Candida, Fusarium and Malassezia. Some relevant bacterial general .
Overall, our observations lend strong support to the concept that mixed microbial infections contribute to or are a risk factor for the neuropathology of PD. Importantly, these results provide the basis for effective treatments of this disease using already approved and safe antimicrobial therapeutics.
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Bratisl Lek Listy. 2006;107(6-7):227-30. Chronic polysystemic candidiasis as a possible contributor to onset of idiopathic Parkinson’s disease.L M Epp, B Mraec
Abstract
The underlying cause of Parkinson’s disease is still enigma. Several mechanisms have been implicated in the etiopathogenesis of PD including oxidative damage, environmental toxins, genetic predisposition, and accelerated aging.
Recent research suggests that salsolinol, a derivate of dopamine, is an important contributing factor. In the presence of acetaldehyde dopamine is converted into salsolinol, a neurotoxin involved in apoptosis of dopaminergic neurons. Increased production of acetaldehyde is associated with chronic polysystemic candidiasis (CPC).
Chronically elevated levels of acetaldehyde in patients with CPC might participate in the formation of salsolinol and its metabolites in the brain contributing to the destruction of dopaminergic cells in substantia nigra. Clinical mental symptoms of PD often correspond with the mental manifestations of CPC.
Robert Rodgers PhD Founder 2004 Parkinsons Recovery robert@parkisonsrecovery.com
The recent studies listed below provide compelling evidence of an association between infections and a diagnosis of Parkinsons. Central nervous infections include Streptococci, Tuberculosis, Lyme disease, Neurosyphilis, spinal abscess and others.
My standard recommendation for individuals dedicated to launch a successful journey down the road to recovery is to determine the cause of symptoms first. Once you know the cause, appropriate therapies can be used to address it.
A formidable complication arises when there are a multiplicity of causes. Infections may not be the only one. Toxins, trauma and limiting thoughts about the prospects of recovery can all be causes along with infections.
Oh my. How in the world do you figure out a viable therapy to treat infections? There are two options:
Work with your doctor to identify which infection needs to be treated.
Support your body to heal from the inside out.
A limitation of the first option is that antibiotics are the standard treatment. The good news is that antibiotics can usually address an infection successfully. The bad news is that they kill the good bacteria as well as the bad in the gut. Given that digestive problems drive symptoms, this is not a welcome outcome.
A limitation of the second option is that while there are many options that nurture healing from the inside out, some will work much better than others for us and everyone is different. Healing from the inside out requires a strict adherence to a toxin free diet. This means no packaged foods, regular exercise and a transformation of beliefs that symptoms are destined to get worse. Traumas that have been embedded at the cellular level also need to be released.
Did you get thoroughly exhausted from reading these limitations? I certainly did. Still, pursing the second option enables the body to heal not only infections, but release toxins and bring a compromised digestive system back on line. Taking a long term strategy which requires patience, commitment and focus will support recovery in the long run.
Since I am a researcher and not a medical doctor, my focus is on the second option which involves helping the body come back into balance and harmony with natural methods and approaches. The body does need extra support when symptoms are problematic.
New therapies have happily emerged in the last few years that offer healing options supported by research studies. These involve the delivery of frequencies through light and sound. Down the road this is unquestionably the medicine of the future.
While the challenge to heal from the inside out is indeed challenging, recent treatment options are making partial or full recovery possible for more and more people with neurological problems. The good news is that more people are celebrating a relief from their symptoms today when compared to 20 years ago.
Recent Studies on Infections and Parkinsons
Neurol Sci. 2026 Jan 6;47(1):110. The spectrum of movement disorders in neurosyphilis: A systematic review
Abstract
Background: Neurosyphilis is a well-recognized mimicker and may present with diverse movement disorders resembling neurodegenerative or autoimmune syndromes. This systematic review aims to elucidate the comprehensive clinical, laboratory, imaging, and outcome spectrum of movement disorders in neurosyphilis.
Methods: This systematic review followed PRISMA guidelines with a protocol registered in PROSPERO (CRD420251135620). PubMed, Embase, Scopus, and Google Scholar were searched. Eligible studies included case reports, case series, and observational cohorts with confirmed or probable neurosyphilis and clinically diagnosed movement disorders. Data on demographics, clinical features, cerebrospinal fluid, neuroimaging, treatment, and outcomes were extracted.
Results: We analyzed a total of 48 individual cases reported across 44 publications and two retrospective cohorts (n = 223, with 61 movement disorder patients). The mean age was 47 years (range 10-83), with 76.3% male and 76.3% human immunodeficiency virus negative. Movement disorder subtypes included ataxia (28.9%), parkinsonism (18.4%), choreiform disorders (15.8%), dyskinesias/dystonia (15.8%), and myoclonus (7.9%). The orofacial “candy sign” was noted in 7.9%. In 10 cases, complex presentations mimicked progressive supranuclear palsy, corticobasal degeneration, Creutzfeldt-Jakob disease, and autoimmune encephalitis. CSF serology was positive in 84.2% of cases. Imaging showed cerebral or cerebellar atrophy (34.2%), basal ganglia infarction (15.8%), white matter changes (15.8%), hydrocephalus (5.3%), or normal scans (18.4%). Penicillin was mainstay of therapy (57.9%). Outcomes included marked improvement in 44.7%, partial recovery in 34.2%, persistent decline in 15.8%, and death in 5.3%.
Conclusion: Neurosyphilis should be recognized as a treatable cause of secondary movement disorders. Ataxia and parkinsonism predominate, but frequent misdiagnosis delays therapy.
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J Med Microbiol. 2026 Mar;75(3):002136. Association between spirochaetal infection and neurodegenerative diseases: a systematic review and quantitative synthesis of observational studies
Abstract
Introduction. Neurodegenerative diseases, including Alzheimer’s and Parkinson’s, are a growing global health concern. While age remains the primary risk factor, infectious agents have been proposed as potential contributors to disease onset or progression.
Spirochaetal bacteria, such as Treponema pallidum, Borrelia burgdorferi and Leptospira spp., can invade the central nervous system, yet the extent to which these infections influence neurodegenerative outcomes remains unclear.
Aim. This systematic review aimed to evaluate observational evidence on the association between spirochaetal infections and neurodegenerative diseases and to identify gaps in the literature to inform future research.
Methodology. A systematic search of SCOPUS, EMBASE, PubMed/MEDLINE, Web of Science and CINAHL was conducted for studies published between January 2000 and May 2025. Eligible studies were observational, involved adult human populations and reported both spirochaetal infection and cognitive or neurodegenerative outcomes using standardized methods. Data were extracted using a standardized form. Owing to heterogeneity in study design, diagnostic approaches, outcome measures and reporting formats, an overall pooled meta-analysis was not feasible; however, a quantitative synthesis using meta-analytic methods was conducted for studies reporting mini-mental state examination data. Risk of bias was assessed using the Newcastle-Ottawa Scale.
Results. Twenty-seven studies met the inclusion criteria: 13 on T. pallidum, 13 on B. burgdorferi and one on Leptospira spp. No eligible studies were found for Brachyspira spp., and studies involving Treponema denticola were excluded due to confounding by periodontitis. Studies investigating syphilis and leptospirosis consistently reported cognitive impairment and increased dementia risk. In contrast, findings for Lyme disease were heterogeneous, with some studies reporting persistent symptoms or increased Alzheimer’s risk, while others found no long-term cognitive effects.
Conclusion. This review highlights a potential link between spirochaetal infections and neurodegenerative outcomes, particularly for syphilis and leptospirosis. Evidence for Lyme disease remains inconclusive.
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ReviewViruses. 2025 Jan 31;17(2):203. Viruses and the Brain-A Relationship Prone to Trouble
Abstract
Neurological disorders, some of which are associated with viral infections, are growing due to the aging and expanding population. Despite strong defenses of the central nervous system, some viruses have evolved ways to breach them, which often result in dire consequences.
In this review, we recount the various ways by which different viruses can enter the CNS, and we describe the consequences of such invasions. Consequences may manifest as acute disease, such as encephalitis, meningitis, or result in long-term effects, such as neuromuscular dysfunction, as occurs in poliomyelitis.
We discuss evidence for viral involvement in the causation of well-known chronic neurodegenerative diseases, such as Alzheimer’s disease, Parkinson’s disease, amyotrophic lateral sclerosis, multiple sclerosis, as well as vascular dementia in the elderly.
We also describe the approaches currently available to control a few of the neural viral infections. These include antivirals that are effective against human immunodeficiency virus and herpes simplex virus, as well as vaccines valuable for controlling rabies virus, poliomyelitis virus, and some flavivirus infections. There is an urgent need to better understand, at a molecular level, how viruses contribute to acute and, especially, chronic neurological diseases and to develop more precise and effective vaccines and therapies.
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Below is a 2012 study of 18,648 patients with Parkinsons that kicked off the interest among researchers in the connection between infections and Parkinsons
Int J Epidemiol. 2012 Apr 19;41(4):1042–1049. doi: CNS infections, sepsis and risk of Parkinson’s disease
Abstract
Background Neuroinflammation may play an important role in the aetiology of Parkinson’s disease (PD); however, little is known about infections in relation to future PD risk.
Methods We conducted a register-based nested case–control study in Sweden to examine infections of the central nervous system (CNS) and sepsis in relation to PD with 18?648 patients and 93?240 matched controls. We defined the index date as the date of first recorded PD diagnosis in the Swedish Patient Register.
Results Overall, PD patients were more likely to have a previous hospitalization for CNS infections [odds ratio (OR)?=?1.5, 95% confidence interval (CI): 1.2–1.9] or sepsis (OR?=?1.6, 95% CI: 1.4–1.7) than controls, largely due to hospitalizations in the year before PD identification (CNS infections: OR?=?3.0, 95% CI: 1.6–5.7; sepsis: OR?=?3.5, 95% CI: 3.0–4.0). However, we found that subjects with multiple CNS infections at least 5 years before the index date had higher PD occurrence than those without CNS infections (OR?=?3.3, 95% CI: 1.4–8.2), whereas the corresponding OR for sepsis was 1.4 (95% CI: 0.8–2.4). After the index date, PD patients were more likely to be hospitalized for CNS infections [hazard ratio (HR) =1.8, 95% CI: 1.2–2.7] or sepsis (HR?=?2.2, 95% CI: 2.1–2.4) than controls.
Conclusions This study provides preliminary evidence for an association between CNS infections, but not sepsis, and a higher future risk of PD. It also shows that PD patients were more prone to CNS infections and sepsis than controls.
When young, we could all eat anything and get away with it. It mattered little that the comfort food we all loved to eat in our youth acidify the body.
Young bodies have the magical ability to buffer acidity. This helps us remain active and happy since an acidic body lowers oxygen levels in the body.
This buffer sadly is just about exhausted by age 45. If we continue to eat the same comfort food diet we loved to eat as a child, our body morphs more cells that extract oxygen from – of all foods – sugar which is the deadliest toxin when it comes to inflaming Parkinsons symptoms.
The good news is that stem cells repair, rejuvenate and regenerate other cells, one among many miracles body performs for us day in and day out. The bad news is that stem cell production peaks at age 25.
This means for everyone – and especially persons struggling with neurological challenges – that if we continue to eat the same comfort foods that gave us such joy when we were young, we will eventually confront one horrible symptom after another. Our body will fight a losing way to keep us healthy and strong.
Why then do so many older people have illnesses of one type of another?
Why is there such a dramatic increase in the number of persons diagnosed with Parkinsons and Alzheimers?
The Answer: Most people are not able to change their diet to one that offers a daily infusion of good bacteria in the form of prebiotics. Let’s face it. Comfort food gives us emotional comfort, even if for only a few minutes, which is hard for anyone to give up.
I personally would like to continue eating foods that I loved eating as a child. But if I continue along this pathway to destruction, I will get sick.
Making the change is indeed difficult. It takes time, commitment and focus to eat healthy food. After all, there was always so much comfort in the foods that gave us such joy when we were young when we never had to worry about the damage it might do.
Those days are over for those over 45 who are determined to live in a healthy body.