In a study published in 2026 posted below, we have still more evidence for the benefits of exercise for individuals experiencing the neurological challenges of Parkinsons. Results of this study found that an eight-week aerobic resistance training program improved tremor and walking endurance in patients with Parkinson’s symptoms.
NeuroRehabilitation. 2026 Apr 10:10538135261434253. Effects of an 8-Week Combined Aerobic Resistance Training on Motor Function, Functional Mobility, and Metabolic Parameters in Patients with Parkinson’s Disease: A Controlled Study
Abstract
Exercise is increasingly recognized as a key component of Parkinson’s disease (PD) management, but data on short-term combined aerobic-resistance training (ART) on motor and metabolic parameters remain limited.
In this controlled study, 28 patients with PD (median Hoehn & Yahr stage= 2, IQR 1.75-2.0) were allocated to an 8-week supervised aerobic-resistance training ART group (n= 19) or a stretching-only control group (n = 9). Training consisted of two 45-minute sessions weekly. Primary outcomes were MDS-UPDRS-III, Sit-to-Stand Test (STS), and Six-Minute Walk Test (6MWT).
Fasting laboratory parameters were exploratory given the small sample size and absence of an a priori power analysis. Between-group post-intervention differences were analyzed using ANCOVA (baseline as covariate); exploratory biochemical outcomes were controlled for multiple comparisons using Benjamini-Hochberg false discovery rate (FDR).
ART improved the MDS-UPDRS-III tremor subscore (p = 0.004) and6MWT distance (p = 0.003), with no significant effects on other motor or functional outcomes. In exploratory laboratory analyses, unadjusted between-group differences were observed for HDL cholesterol (p =0.014), vitamin D3 (p = 0.003), TSH (p = 0.042), and homocysteine (p =0.019); after FDR correction, only vitamin D3 remained significant (qFDR= 0.039). Total cholesterol, LDL, and VLDL showed non-significant decreases.
An eight-week ART program improved tremor and walking endurance inpatients with PD, while other motor outcomes remained unchanged. Exploratory changes in selected biochemical markers were detected (including a decrease in vitamin D3).
The truthful answer to the question “why is Parkinson’s disease increasing” will likely surprise you. Is it because of the increasing toxins in our environment? Not really, but that is certainly a factor. Is it because of contaminated water systems? Not really, but that too is a factor. Enough. The answer is …
If you watched my video, you were likely surprised at my answer. There is typically one and only one answer to the question – what causes Parkinson’s disease. The answer? A lack of dopamine. Everyone knows that, duh.
Take for example the Parkinson’s disease hand tremor. When the right medications or supplements and the right dose for your body that contain L-dopa are taken. the hand tremor is quieted. The L-dopa is converts to dopamine in the body. So maybe the reason Perhaps the answer to Why is Parkinson’s disease is increasing turns on the decreasing supply of L-Dopa?
End of story, right? I say wrong. Parkinson’s disease is caused by a persistent fear people cannot not shake. The fear is familiar to us all: Lack of control over everything and everyone.
Dynamics of the world spin so rapidly these days many people feel they do not control anything – not their pay check, not the addictions of family members, not the untimely deaths of family and friends, not global warming, not …
In the video I explain how to turnaround the feeling of being out of control with an example of how to react when Parkinsons disease hand tremors become problematic. Do we sit on our hands? Do we tuck them in our pocket? There is another approach.
In summary – More and more people are feeling they are out of control which is the simple and obvious reason why Parkinsons disease is increasing.
Twenty years of research and interviews with persons diagnosed with Parkinsons have unearthed a rich set of natural therapies to reverse symptoms. These discoveries are documented in my book Road to Recovery from Parkinsons Disease 2026.
This 2026 book includes the therapies and methods that have enabled others with Parkinsons to celebrate a successful journey down the road to recovery.
Just released for the price of one mocha cafe a week … everything you need to know about the causes of Parkinsons and heir treatments to manifest a successful recovery from Parkinsonsin 2026
* A new audiobook clip posted every day * Comprehensive review of causes * Coverage of therapies to treat specific causes * Overview of how successful recoveries are manifested * Proven formulas for success * Daily support that transform negative thoughts into positive ones * Cutting edge therapies that are transforming recovery in 2025 * Low monthly tuition during these challenging economic times. * Cancel anytime.
Daily recordings report what founder of Parkinsons Recovery Robert Rodgers PhD has discovered over two decades of research exclusively focused on Parkinsons. Visit the link below to learn more and register.
Two L-dopa sources for Parkinson’s disease dopamine are the prescription medication Sinemet and the over the counter supplement Mucuna. Compounding Pharmacist Randy Mentzer answers the following question:
“Can both be taken and if so can there be unwanted side effects?”
Treatment of Parkinson’s disease symptoms often involves taking medications aimed at increasing Parkinson’s disease dopamine levels in the body. Among the various treatment options, Sinemet (a combination of levodopa and carbidopa) is prescribed by doctors to help alleviate motor symptoms by replenishing dopamine.
Patients and practitioners sometimes add mucuna pruriens, a natural supplement, to the treatment regimen. While mucuna does not require a prescription from a doctor it is, like Sinemet, a medicine that contains L-dopa. The body converts the L-dopa to dopamine.
Understanding Sinemet
Sinemet is a prescribed medication for Parkinson’s disease that combines two drugs:
Levodopa: This precursor to dopamine is converted into the neurotransmitter dopamine in the brain, which is crucial for regulating movement and coordination.
Carbidopa: This component prevents levodopa from being converted into dopamine before it reaches the brain, which helps to maximize the effectiveness of levodopa and reduce side effects like nausea.
While Sinemet is effective, its impact usually diminishes over time. Patients usually need to take higher and higher doses to achieve the desired result.
Enter Mucuna Pruriens
Mucuna pruriens, often referred to as velvet bean, is a natural source of levodopa. It’s been used in traditional medicine for centuries and is now gaining attention in the context of Parkinson’s disease treatment. The beans of the mucuna plant contain L-dopa, the same compound found in Sinemet.
Potential Benefits of Combining Mucuna with Sinemet for Parkinson’s Disease Dopamine
Enhanced Dopamine Levels: Since mucuna contains l-dopa, it may potentially boost dopamine levels in the brain, complementing the effects of Sinemet. Some studies suggest that mucuna could offer a more rapid or sustained increase in dopamine levels compared to synthetic levodopa.
Reduced Motor Fluctuations: Mucuna might help in smoothing out the “on-off” fluctuations experienced by some Parkinson’s patients on Sinemet alone. The natural levodopa in mucuna might work more consistently to help manage symptoms throughout the day.
Possible Reduction in Side Effects: Some users report fewer side effects with mucuna compared to synthetic levodopa preparations. This could be due to the natural formulation of mucuna.
Considerations and Risks
Lack of Standardization: Unlike prescription medications, the potency and purity of mucuna supplements can vary widely. This lack of standardization means that the exact dose of levodopa can be unpredictable.
Drug Interactions: Mucuna may interact with Sinemet, potentially altering the effectiveness of either medication. It’s crucial to monitor any changes in symptoms and adjust dosages under medical supervision.
Side Effects: While mucuna might have fewer side effects for some, it’s not without risks. Potential side effects include dyskinesia (the side effect Randy mentions in his answer to the question posed to him above), nausea, dizziness, or digestive issues. Always discuss these possibilities with your healthcare provider.
Practical Tips for Combining Mucuna with Sinemet
Consult Your Doctor: Before introducing mucuna into your regimen, have a thorough discussion with your neurologist or primary care physician. They can provide personalized advice based on your specific condition and treatment goals.
Now here is something interesting to do, especially if you ever wondered what your voice says about you. But first – what is voice profiling?
Sound Health has discovered that the frequencies contained in the voice are holographic representations of your state of health and wellness. Every muscle, compound, process and structure of the body has a Frequency Equivalent™ that can be mathematically calculated. It is our belief that people with similar trauma, illnesses, syndromes, psychologies, diseases, toxins… have similar, if not identical, vocal anomalies.
You can obtain an analysis of your voice profile which is available on the Sound Health Portal for free. Here is the link to get a free analysis of your voice.
Once you land on the Nano Voice page above, click Start. Then, talk in a normal voice without pauses for 90 seconds. An analysis of the frequencies emitted by your voice will be instantly provided.
Once you generate your first overview report, you can restart Nano voice and talk a second time for 90 seconds to generate a follow-up report. I talked for 90 seconds three times back to back. The three reports of my personality were all very similar. I must report they also described me to a tea.
Why bother?
It is free
It takes only 5 minutes
The instant report that is generated is fascinating
This short activity will convince you to take analysis of voice profiles seriously. Healing through sound (and light) is the medicine of the future
What does it take to reverse symptoms with Parkinson’s? I have documented what individuals have done to celebrate a successful recovery and captured their wisdom in a Parkinsons Recovery® program that offers daily support for recovery from Parkinsons.
Have the solutions been simple?
Were they straightforward??
Was there a particular therapy that accounted for the success?
Was success quick?
When I review the profile of the dozens of success stories, the answer to all of these questions is no.
Solutions are not simple or straightforward.
Multiple therapies and approaches were needed.
Recovery required experimentation to discover what worked and what did not.
Solutions are not simple or straightforward.
Multiple therapies and approaches were needed.
Recovery required experimentation to discover what worked and what did not.
Daily support for recovery from Parkinsons is now available to support your personal journey down the road to recovery. Join others who have succeeded in reversing their symptoms.
Homocystene is an amino acid that circulates in the blood. It is produced when the body breaks down protein. Low homocysteine levels are considered healthy. Elevated levels are problematic for a number of reasons. High levels …
Are a marker of inflammation
Cause dementia
Can cause strokes
Damage artery walls
Increase blood clot risks
Compromise the cardiovascular system.
A reason homocysteine levels can be too high in the case of Parkinsons is that certain medicines delete vitamins B6, B12 and folic acid (vitamin B9). How do you reduce unusually high levels of homocysteine? Increase the intake of vitamins B6, B12 and folic acid – the ones that just happen already to be chronically deficient.
Supplements of course can be taken to supplement low levels of B vitamins. Alternatively, certain foods boost the level of B vitamins. The following foods are rich in vitamins B6, B12 and folic acid:
Spinach
Beef liver
Chicken liver
Chickpeas
Sunflower seeds
Wild salmon
Eggs
Clams
Oysters
Sardines
Lentils
Research on Homocysteine Levels and Parkinsons
Heliyon. 2024 Nov 6;10(21):e40191. Association analyses of nutritional markers with Parkinson’s disease and Alzheimer’s disease
Abstract
Introduction: Parkinson’s disease (PD) and Alzheimer’s disease (AD) are common neurodegenerative diseases with multifaceted etiology. Nutritional and metabolic abnormalities are frequently implicated in PD and AD. In this observational study, we analyzed a series of nutritional markers, and aimed to understand their association with AD and PD risk.
Methods: A total of 424 PD patients, 314 AD patients, and 388 healthy controls were included. Nutritional markers including Hemoglobin A1c, vitamin B12, folate, apolipoprotein B (ApoB), apolipoprotein A1 (ApoA1), low-density lipoprotein (LDL), high-density lipoprotein, triglyceride, total cholesterol (TC), uric acid and homocysteine (HCY) were measured. Significance for odds ratios examining was P < 0.0045 after bonferroni correction.
Results: Multifactor risk analysis showed that ApoB, LDL, and TC reduce PD risk, while homocysteine HCY increase PD risk. ApoA1 and HCY are protective and risk factors for AD, respectively.
Conclusion: The cross-sectional study demonstrates that HCY and lipid metabolism markers are associated with PD and AD risks. Our findings support the involvement of one-carbon metabolism and lipid metabolism disturbance in PD and AD.
Nutrients. 2025 Apr 11;17(8):1325. Homocysteine, Nutrition, and Gut Microbiota: A Comprehensive Review of Current Evidence and Insight
Abstract
Homocysteine, a sulfur-containing amino acid, is an intermediate product during the metabolism of methionine, a vital amino acid. An elevated concentration of homocysteine in the plasma, named hyperhomocysteinemia, has been significantly related to the onset of several diseases, including diabetes, multiple sclerosis, osteoporosis, cancer, and neurodegenerative disorders such as dementia, Alzheimer’s and Parkinson’s diseases.
An interaction between metabolic pathways of homocysteine and gut microbiota has been reported, and specific microbial signatures have been found in individuals experiencing hyperhomocysteinemia.
Furthermore, some evidence suggests that gut microbial modulation may exert an influence on homocysteine levels and related disease progression. Conventional approaches for managing hyperhomocysteinemia typically involve dietary interventions alongside the administration of supplements such as B vitamins and betaine.
The present review aims to synthesize recent advancements in understanding interventions targeted at mitigating hyperhomocysteinemia, with a particular emphasis on the role of gut microbiota in these strategies. The emerging therapeutic potential of gut microbiota has been reported for several diseases.
******
.Acta Neurol Belg. 2025 Apr;125(2):435-444. Diagnostic values of serum levels of ?-synuclein, homocysteine and leucine-rich ?2 glycoprotein for predicting cognitive impairment in Parkinson’s diseas
Abstract
Background: ?-synuclein, homocysteine (Hcy) and leucine-rich ?2-glycoprotein (LRG) have been shown to correlate to Parkinson’s disease (PD). However, it remains unclear whether these factors are associated with the occurrence of cognitive impairment (CI) in PD patients.
Methods: This study initially enrolled eligible PD patients without cognitive impairment. Blood samples were collected to measure serum levels of ?-synuclein, Hcy, and LRG using enzyme-linked immunosorbent assay. After one year of treatment, patients were divided into CI group and non-CI groups based on their Montreal Cognitive Assessment (MoCA) scores. Baseline clinical characteristics and the levels of these three factors were compared between the two groups. Additionally, Receiver Operating Characteristic (ROC) analysis was used to assess the predictive value of these factors for the occurrence of CI in PD patients.
Results: The study included 102 eligible PD patients without baseline CI, divided into 65 patients in the non-CI group and 37 patients in the CI group after one year, based on the MoCA scores. Serum levels of ?-synuclein, Hcy, and LRG in the CI group were 0.42 ± 0.33 ng/mL, 19.85 ± 6.31 µmol/L, and 14.53 ± 5.11 ng/mL respectively, compared to 0.04 ± 0.03 ng/mL, 14.32 ± 5.25 µmol/L, and 11.67 ± 3.10 ng/mL in the non-CI group. Patients in the CI group had MoCA scores below 26, indicating cognitive impairment. ROC analysis revealed that ?-synuclein, Hcy, and LRG levels effectively predicted the occurrence of CI in PD patients.
Conclusions: Serum levels of ?-synuclein, homocysteine and glycoprotein were elevated in the CI group compared to the non-CI group, suggesting that these factors may serve as predictors of cognitive impairment in PD patients.
A 10 percent discount is available with coupon code healing4me. If your child does not show the improvement anticipated after 6 months of daily use, you can return the Duo device for an 80% refund
Ground Breaking Discovery for Autism Using the Vielight Neuro Duo
As the world’s only transcranial-intranasal brain photobiomodulation device, the Vielight Neuro Duo is the result of years of engineering and research.
Groundbreaking discovery for autism research with the Vielight technology produced improvements in cognition, memory and blood flow.
The Vielight Neuro Duo comes with both Alpha and Gamma modes.
Gamma (40Hz): focus, memory, brain energy.
Alpha (10 Hz): relaxation and sleep improvement.
Summary for the Autism Spectrum Disorder Clinical Study
Children (BASEL). 2022 May 20;9(5):755. Transcranial Photobiomodulation for the Treatment of Children with Autism Spectrum Disorder (ASD): A Retrospective Study
Data were reported and analyzed for 21 children, 13 males and 8 females with an average age of 9.1. The age range is 5 to 15 years old. The study was conducted over six months in the home setting.
Participants used the Vielight Neuro Duowith its two protocols, the Alpha 10 hertz protocol and the Gamma 40 hertz protocol.
Both Alpha and Gamma protocols were administered each day, one in the morning and one in the evening. Each session had a duration of 20 minutes, during which children were involved in stimulating activities such drawing, coloring, reading, playing games or doing homework.
Findings
After 6 months, transcranial-intranasal photobiomodulation was associated with:
reduction in ASD severity, as shown by a decrease in CARS scores (p < 0.001).
reduction in noncompliant behavior
reduction in parental stress
reduction in behavioral and cognitive rigidity
improvement in attentional functions and in sleep quality.
These remarkable improvements not only enhanced the lives of the patients but also greatly reduced stress among their parents, as measured by the SDAG scale, creating a more peaceful and effective environment for growth.
How Photobiomodulation Reverses Austism
How does the Vielight Neuro Duo photobiomodulation device work? It emits an optimal irradiance, amongst the highest in the industry with the 810 nanometer wavelength to penetrate the skull and reach the brain, transcranially and intranasally. The Vielight Neuro Duo’s design specifically targets the Default Mode Network to improve connectivity between its nodes.
The default mode network is crucial in understanding autism. Individuals with autism spectrum disorder have been observed to have alterations in their default mode network, contributing to difficulties in social interactions and understanding others’ perspectives.
Research has revealed that atypical connectivity within the default mode network underlies the characteristic challenges in social communication and repetitive behaviors seen in autism.
The two different pulsing modes, Alpha and Gamma have complementary effects. The alpha protocol promotes relaxation, reducing rigidity and improving sleep. The gamma protocol boosts cognitive functions which facilitates improved focus and attention.”
Safety and Future Directions
From a safety perspective the treatment has been well-tolerated with minimal side effects, making it a safe option.
The second study abstract below reports positive results for younger children ages 2-6.
Front Neurol. 2024 Apr 26:15:1221193. Transcranial photobiomodulation in children aged 2-6 years: a randomized sham-controlled clinical trial assessing safety, efficacy, and impact on autism spectrum disorder symptoms and brain electrophysiology
Abstract
Background: Small pilot studies have suggested that transcranial photobiomodulation could help reduce symptoms of neurological conditions, such as depression, traumatic brain injury, and autism spectrum disorder.
Objective: To examine the impact of photobiomodulation on the symptoms of Autism in children aged two to six years. (The study listed above is based on older subjects 5-15 years in age).
Method: This study ran a randomized, sham-controlled clinical trial involving thirty children aged two to six years with a prior diagnosis of ASD. Pulses of near-infrared light (40 Hz, 850 nm) noninvasively were delivered to selected brain areas twice a week for eight weeks, using an investigational medical device designed for this purpose. We used the Childhood Autism Rating Scale (CARS) to assess and compare the Autism symptoms of participants before and after the treatment course. Electroencephalogram data during each session were collected from participants who tolerated wearing the cap.
Results: The difference in the change in CARS scores between the two groups was 7.23 (95% CI 2.357 to 12.107, p = 0.011). Seventeen of the thirty participants completed at least two EEGs and time-dependent trends were detected. In addition, an interaction between Active versus Sham and Scaled Time was observed in delta power (Coefficient = 7.521, 95% CI -0.517 to 15.559, p = 0.07) and theta power (Coefficient = -8.287, 95% CI -17.199 to 0.626, p = 0.07), indicating a potential trend towards a greater reduction in delta power and an increase in theta power over time with treatment in the Active group, compared to the Sham group. Furthermore, there was a significant difference in the condition (Treatment vs. Sham) in the power of theta waves (net_theta) (Coefficient = 9.547, 95% CI 0.027 to 19.067, p = 0.049). No moderate or severe side effects or adverse effects were reported or observed during the trial.
Groundbreaking Discovery for Autism Conclusion:
Findings of these two studies taken together indicate that photobiomodulation is a safe and effective treatment for Autism. The Vielight Neuro Duo offers a groundbreaking treatment for Autism.
A 10 percent discount is available with coupon code healing4me. If your child does not show the improvement anticipated after 6 months of daily use, you can return the Duo device for an 80% refund.
Call Vielight to get answers to your questions. The company is located in Toronto Canada: 1-877-355-8012 (Main line)
One dominant source of toxins for persons experiencing neurological challenges is polluted drinking water. One option many people embrace is to filter the water that they drink. This is a marked improvement over drinking unfiltered tap water. But what about distilled water? I this video I offer a comparison of distilled vs filtered water.
My honest confession is that I had never considered distilled water until a few months ago when it was recommended by our naturopath doctor. My initial reaction was to wonder whether bothering to distill water every day was really worth the effort.
What is Distilled Water?
Distilled water involves boiling it into steam, then condensing the steam back to a liquid. This removes toxins, bacteria, chemicals and microbes. It does take 5-6 hours to boil a gallon of water to make distilled water. Is the bother worth it?
As I point out in the video, I am shocked every day at the residue of gunk, sludge and goop that remains in the container after the gallon of water is distilled. Oh my. I have been putting that sludge into my body all this time (which has been a few decades).
Admittedly some of the sludge that remains consists of beneficial substances like minerals. But most of the gunk is toxic. I would prefer to get the minerals my body needs from eating nutritious food rather than drinking water that is laced with toxins.
There are many distilled devices on the market. Here is the one that we have been using.
I have a heads up on the most fascinating interview below. Leif Ogard has had Parkinsons for over two decades. Leif has published a book with the quite fascinating title of I Have Parkinsons But Parkinsons Does Not Have Me.
Leif feels great – not just good – but great. Many people who do not experience symptoms of Parkinsons cannot say this! He has incredible energy and owns his own business which he started after his diagnosis.
By the way, his business is thriving. Leif considers himself healthy in every respect.
In this interview Leif offers genuinely helpful insights and suggestions. His perspectives will be especially helpful for anyone who has been recently diagnosed.
One of the testimonials for his book posted on Amazon reads:
“Leif Ogard is an engineer and consultant by profession. His meticulous attention to detail about his condition, life and attitude are inspirational. He over came his own fears and self doubts to accomplish extra ordinary feats and still does). He decided rather than be a victim, he would control his life by studying, documenting and controlling everything that influenced his body.”
If you are in a place in your life where you could use a motivational boast listen to my interview with Leif. You will not be disappointed. on Blog Talk Radi
The cover of Leif’s book is shown here. Believe me when I tell you that his life is true to the title of his book.
“As the middle of July approaches I am intensely aware that almost 1 year ago my husband was diagnosed with Parkinsons Disease. I reflect back on this deeply emotional year and am actually surprised at some of our learning and growth plus the continuous challenges. And interestingly, I am also aware of a new sense of joy and intimacy in our relationship.
But our year did not begin that way. I go to a school of healing. One that teaches that energy follows thought and physical illness may be a result of our beliefs. Through our physical illnesses we have an opportunity to heal ourselves in the areas of our lives where we have previously been stuck or have held unconscious negative thoughts about ourselves and our lives.
I was very aware that in my school the line of thought may be, ‘What is it your husband needs to learn from having Parkinsons?’ and indeed a classmate did ask me that, although in a very loving and concerned way. What are his beliefs about Parkinsons?
I had read the book by Louise Hay, You Can Heal Your Life where she correlates probable beliefs about Parkinsons to fear and an intense desire to control everything and everyone. Another author, Karol Truman, adds to this with several more possibilities: lack of inner communication, fear of not being able to control and not understanding a fear you have which stops you from believing in what you do and say.
I gently asked my husband if any of these feelings or beliefs resonated for him but nothing did at the time. As an outside observer, I was wondering about the possibility of the lack of communication with his inner or real self.
As the year unfolded, a big struggle for my husband was what to say when people talked to him about Parkinsons Disease. Someone once said to him very sincerely, ‘I am sorry about your diagnosis’ and my husbands response was to laugh.
I discussed this with him and suggested that laughing may have been a way not to feel a deeper emotion. We also talked about how his response to illness is an opportunity to teach others and help them to connect with their own vulnerabilities, insecurities and feelings about illness.
The other day we were invited to a party and my husband was nervous that Parkinsons would be discussed. We decided to do some role playing to help him come up with some responses.
I said to him (as someone previously did to me), ‘You know, people with Parkinsons do not have the tremor when they sleep. He replied, ‘Oh, have you slept with someone with Parkinsons? I burst out laughing. Yes, that would be a good response to that.
Then I asked him what his plans were for the future now that he had Parkinsons and he burst out laughing. ‘I am sorry’, he replied. ‘It is just too serious a question not to laugh.
Then I got a little tougher. I said, ‘I am sorry about your diagnosis’ and he very quietly and gently said, ‘I am sorry too.
My fiancee was diagnosed with Parkinson’s 3 years ago.
We both would like to explore the best Parkinsons natural remedies. To make a long story short, he’s made many lifestyle changes (better dietary choices; no chemicals in his home; takes soil-based organisms; limits stresses…) and has been to multiple ND’s as well as MD’s for interventions.
He’s only tried one conventional medicine, Reserp, which resulted in severe palpitations. He has refused all other medications because he believes that this only addresses the symptoms and not the cause.
This is only a brief synopsis of what’s transpired over the past few years, but I’m hoping I’ve included enough info for you.
What can we try to help him recover from this disorder? Neither of us believe that this “disease” is incurable, but we are at a loss as to what to try next. Any help you can send my way would be MOST APPRECIATED.
You have asked a great question. Parkinsons Recovery
is all about identifying a wide variety of natural alternatives
for people to consider who are committed to feeling
better.
You fiancee clearly is on the road to recovery and he
will get better. Keep in mind two realities. First, recovery
is a gradual process which takes time and patience.
You don’t start eating healthy food one week and see
dramatic improvements the next week. It takes the body
time to rejuvenate cells and to heal.
Second, you do not report whether he is seeing any relief
from symptoms. Is he getting worse? Is he the same? Is
he getting better? My guess is that you and he probably
do not know the answer
From the tone of your letter is sounds like you are
both discouraged. It is very possible that he is
actually improving – but neither of you see it. No one
is adept at tracking improvement in symptoms. We
all tend to focus on the problems not the successes.
You use the terms “better dietary choices” which is
an interesting use of terms. You do not say “healthy
dietary choices.” It is possible that while his eating habits are
generally good he may be eating foods he is
allergic to. Keep in mind also that sugar is a
neuro toxin. If he is eating any sugar at all
it will be a challenge to celebrate symptoms relief.
You say he has no chemicals in his home. That
is awesome news. He may however have certain
toxins lodged in the tissues of his body. Most people
do. It is just a question of whether the extent of
exposure is too much for his body to process.
You do not mention hydration. When the body is
adequately hydrated it is able to cleanse itself
of toxins more readily. Naturopath John Coleman recommends
a homeopathic remedy for dehydration called the
aquas. You can find more information at www.aquas.us
I would venture a guess that a factor
that is aggravating his symptoms is a sustained
and persistent levels of stress and trauma. You say
he “limits” stress, but the truth is that any stress
can have a profound impact on our immune system
and on the health of our neurological system.
So as a final next step to consider, I would recommend
he investigate methods of releasing stress and trauma
that is lodged in the tissues of his body. As long as a
body is in a sustained state of stress the symptoms
will not and cannot resolve. Exploring ways to release
the trauma that is trapped at the cellular level should
rise to the top of the list of Parkinsons natural remedies..
There are many awesome approaches for relieving
stress and trauma that is lodged in the tissues of
the body: craniosacral therapy, Bowen therapy,
neurolinguistic programming, biofeedback
to mention just a few.
Which approach is right for him? Only he can figure that
answer out through experimentation using his intuition.
Different therapies work for different people. Only he
will know what is right for him.
I hope these suggestions are of some use. Keep in
mind that there are dozens and dozens of therapies that
have helped people with Parkinson’s. Your fiancee’s job
now is to figure out the ones that are a perfect fit for him.
He has set his intention. He has engaged the journey.
He will eventually get better as he and you experiment with various
Parkinsons natural remedies. Just watch. Be sure to celebrate
his progress as the two of you together track the
improvement in his symptoms over time.
What about Antioxidants and Parkinsons disease? Is Noni Juice helpful as a treatment for symptoms of Parkinson’s?
Paul
Response
There is a very long list of symptoms that have been linked to a diagnosis of Parkinson’s Disease. In fact – if you examine and analyze this list of symptoms, you will cover just about any diagnosis imaginable.
I am aware of no research that directly evaluates the use of Noni juice on the symptoms of Parkinsons, but this is the norm. Little formal evidence exists that evaluates natural treatments.
So What about Antioxidants and Parkinsons Disease?
Certainly, anti oxidants could potentially lend relief to some of the symptoms. We know that anti oxidants help the body reverse the process of aging. To the extent that aging is associated with a degradation of the neurological system, it seems reasonable to suspect that you should be able to expect improved health on some level with the ingestion of anti-oxidants.
Be aware that there are many choices you can make with regard to the use of anti oxidants. A search on the Internet will list for you foods that have natural anti oxidant properties (like blue berries for example).
The decision to take Noni juice or some other anti oxidant hinges on cost and personal preference. I believe everyone – regardless of whether they currently experience the symptoms of Parkinsons or not , should be taking anti-oxidants of some form or another.
In this amazing video, Sharry Edwards from Sound Health Options explains how voice profiling determines causes of Parkinsons. Her approach has revealed fascinating insights into the various reasons people develop symptoms of Parkinsons disease.
This presentation follows up voice profiles Sharry did for four guests on one of my Parkinsons Recovery radio shows last month. Sharry identifies surprising commonalities across the persons she profiled during the show.
Even more interesting is her analysis of the reason Michael J. Fox currently experiences the symptoms of Parkinson’s. Are you at all curious about the cause of his symptoms? You can get the answer by watching this video.
Sharry takes you through the process of voice profiling step by step to demonstration how voice profiling determines causes of Parkinsons. She also offers an amazing explanation of the many reasons symptoms can evolve and what you can do about them when they do.
What are two simple suggestions to support recovery from Parkinsons?The vast number of natural therapies documented by Parkinsons Recovery that support recovery is indeed overwhelming. So many choices! Which ones offer the greatest possibility of relief for you?
The challenge for everyone is that no option – whether a prescription medicine, surgery or natural treatment helps everyone celebrate recovery. Most people celebrate relief from symptoms for many of the options but not everyone. It comes down to the challenge of deciding which treatment options are most likely to benefit you.
Two Simple Suggestions to Support Recovery
I asked Dr. Bruce Fife, ND, to offer two (and only two) suggestions among the many that are available that he suggests would be most likely to benefit the most people. Here is his unexpected answer:
First, add coconut oil to your diet. Start with 1 tablespoon a day added to food. Gradually Increase to 5 tablsspoons a day.
Second, adopt a low carb diet. With Parkinsons the brain has difficulty processing glucose. Ketones are the much preferred food for the brain.
Might I add that the cost of these two suggestions is minimal relative to the anticipated benefit. I say go for it!
Pioneers of Recovery from Parkinsons offer rich insights into what it takes to celebrate relief from Parkinson’s symptoms. The ten individuals featured in discovered different ways to reverse their Parkinson’s symptoms.
Pioneers of Recovery from Parkinsons print book is available on Amazon here: Pioneers of Recovery
Your download is not automated. It usually takes a few hours to receive it in your email or the next day it you order during the evening.
About Pioneers of Recovery from Parkinsons
Below are minute long clips from the Pioneers of Recovery who offer rich insights into their recovery process. Causes of their symptoms varied considerably. Their approaches to recovery were all very different.
Bianca on Recovery
Cheryl on Recovery
Daniel on Recovery
Gord on Recovery
Howard on Recovery
Karl on Recovery
Lexie on Recovery
Lydia on Recovery
Marian on Recovery
Discover the details of how each pioneer reversed their symptoms which are documented in Pioneers of Recovery available on Amazon.
Or better yet, listen to my interviews with each of the pioneers. You will be inspired by the many options that make recovery possible
Studies published in 2026 pinpoint the value of a curumin treatment for Parkinsons.
What is Curcumin?
Curcumin is a spice used often in Indian, Asian and Middle Eastern cuisines for its earthy earthy flavor. Added often to curries, stews, rice and marinades. It is also widely used in health-focused cooking for its anti-inflammatory properties.
It is actually an extract from Turmeric which is also a flavorful spice that offers gentle detoxes when added to food. Turmeric contains a variety compounds. Curcumin is one of those compounds with health benefits because of its antioxidant and anti-inflammatory properties. Turmeric contains only up to 8% curcumin which makes curcumin supplements and spices far more concentrated than Tumeric.
I received the following email reporting positive results from using curcumin to address dementia challenges.
Anyone out there using turmeric or curcumin with good results? We’re trying this spice that crosses the blood brain barrier, giving 200 mg to 700 mg at least 3x per day for dementia/Alzheimer. So far, seeing some good results.
Studies Published in 2026 Finding Benefits of Curcumin Treatment for Parkinsons
Front Pharmacol. 2026 Mar 9:17:1779921. Pharmacological effects and mechanisms of curcumin in animal models of Parkinson’s disease: a systematic review and meta-analysis
Abstract
Background: Curcumin has been demonstrated to possess promising neuroprotective potential in Parkinson’s disease; however, its overall effects remain inconclusive, and its multiple mechanisms of action have not been systematically summarized.
Objective: This systematic review and meta-analysis aimed to evaluate the pharmacological effects of curcumin in animal models of Parkinson’s disease and to investigate its potential mechanisms involving antioxidant, anti-inflammatory, and neuroprotective effects, thereby providing a theoretical basis for its potential clinical application in Parkinson’s disease.
Methods: A comprehensive search of four databases (EMBASE, PubMed, Web of Science, and the Cochrane Library) up to August 2025 identified 31 eligible studies involving a total of 552 animals. Methodological quality was assessed using the SYRCLE risk of bias tool. Standardized mean differences (SMDs) were calculated to evaluate the effects of curcumin on motor function, neurochemistry, inflammation, and oxidative stress in animal models of Parkinson’s disease.
Results: The results demonstrated that curcumin intervention improved motor function in animal models of Parkinson’s disease, as evidenced by increased locomotor distance in the open field test (SMD = 1.25) and elevated mean velocity (SMD = 1.42), prolonged latency to fall in the rotarod test (SMD = 2.49), shortened descent time in the pole test (SMD = -1.16), and reduced traversal time on the balance beam (SMD = -2.27). Curcumin exhibited neuroprotective effects through increasing the number of tyrosine hydroxylase-positive cells (SMD = 2.12), maintaining dopamine levels (SMD = 4.11), and elevating 3,4-dihydroxyphenylacetic acid concentrations (SMD = 3.15). Regarding anti-inflammatory effects, curcumin significantly reduced multiple inflammatory markers, including interleukin-6 (SMD = -4.73), interleukin-1? (SMD = -3.30), tumor necrosis factor-? (SMD = -3.19), and nitric oxide (SMD = -4.91). With respect to antioxidant activity, curcumin significantly reduced malondialdehyde levels (SMD = -4.69) while increasing the activities of superoxide dismutase (SMD = 3.90), glutathione (SMD = 2.08), and catalase (SMD = 2.00).
Conclusion: Curcumin demonstrates significant neuroprotective effects in Parkinson’s disease animal models, improving motor deficits and neuronal integrity likely through multi-target mechanisms involving anti-inflammatory and antioxidant pathways.
******
Drug Des Devel Ther. 2026 Jan 22:20:566113. Therapeutic Efficacy of Curcumin Nanoparticles in Parkinson’s Disease: An Integrated Analysis of Network Pharmacology, Experimental Validation, and Gut Microbiota
Abstract
Background: Parkinson’s disease (PD) ranks as the second most common neurodegenerative condition globally, with a rising occurrence alongside the aging demographic. Previous studies have demonstrated that curcumin monomers can alleviate PD symptoms and slow disease progression, whereas nano-drug delivery systems significantly improve their bioavailability. Notably, gut microbiota imbalance significantly contributes to the onset and advancement of PD; however, the mechanisms by which curcumin nanoparticles produce their therapeutic impact on PD are not yet well understood.
Materials and methods: Initially, network pharmacology was tapped to forecast the probable targets and signaling routes for curcumin in combating Parkinson’s Disease. Later on, molecular docking techniques and molecular dynamics experiments were utilized to substantiate its binding effectiveness. Curcumin nanoparticles were synthesized via the emulsion-solvent evaporation technique. After establishing PD models, multidimensional validation was performed using behavioral experiments, serological assays, Western blotting, hematoxylin-eosin staining, and 16S ribosomal RNA (16S rRNA) sequencing.
Results: Network pharmacology analysis revealed that curcumin acts through multiple targets and pathways. In vivo experiments demonstrated that curcumin nanoparticles enhanced motor capabilities in Parkinson’s disease mice, bolstered antioxidant enzyme levels, alleviated oxidative stress, and inhibited neuronal apoptosis via the Akt signaling pathway. Histopathological analysis showed significant improvements in the number and arrangement of neurons in the hippocampal dentate gyrus (DG) region. Furthermore, remodeling of the gut microbiota and metabolic regulation alleviated neuroinflammation and enhanced neuroprotection.
Conclusion: Curcumin nanoparticles inhibit neuronal apoptosis by activating the Akt signaling pathway and, while also remodeling the gut microbiota microenvironment, collectively ameliorate PD pathology. This study provides pharmacological evidence that curcumin nanoparticle therapy mediates its therapeutic actions through multiple mechanisms, and identifies potential therapeutic targets for PD treatment.
Posted below is the fascinating article – Parkinson’s: A BioAcoustic Perspective – written by Sharry Edwards from Sound Health Options. The article presents findings from the BioAcoustic analysis for three individuals diagnosed with Parkinsons. This analysis reveals three very different causes.
Sharry’s research over the past several decades has identified more than a set of three factors that cause symptoms of Parkinsons. The Sound Health Options list of causes is quite extensive indeed. It includes among others …
Allergies – especially to milk protein and gluten
Lyme disease
Peripheral neuropathy due to the measles virus
Tetanus
Inability to use glycine
Lack of glutathione
Stiff man syndroms
Lack of receptors for some amino acids
Trauma
Emotional
Sharry uses analysis of voice profiles to determine causes. For more information about her revolutionary and groundbreaking approach and to obtain an anlysis of your voice profile visit:
Or simply call her office which is located in Albany Ohio: 740-698-9119
Below is the article she recently published:
Parkinson’s: A BioAcoustic Perspective: Breaking the Sound Barrier of Parkinson’s Disease by Sharry Edwards
Abstract
Parkinson’s disease is traditionally defined as a neurodegenerative disorder characterized by dopamine depletion and motor dysfunction. However, emerging evidence—supported by BioAcoustic vocal analysis—suggests that Parkinson’s may not be a singular disease entity, but rather a collection of distinct biological breakdown patterns that converge into similar symptoms.
This paper presents three case-based profiles demonstrating fundamentally different underlying mechanisms of Parkinson’s, as identified through frequency-based vocal analysis. These findings suggest a paradigm shift: Parkinson’s may be better understood as a pattern of expression rather than a single disease, requiring individualized identification and intervention.
Introduction: The Limits of a Single Diagnosis
Parkinson’s disease has long been associated with:
Loss of dopaminergic neurons
Tremors, rigidity, and slowed movement
Progressive neurological decline
While these symptoms are consistent, the cause is not.
Patients with identical diagnoses often respond differently to the same treatments. This inconsistency suggests that Parkinson’s may arise from multiple biological pathways—each requiring a different approach.
BioAcoustic Biology proposes that the human voice is a real-time representation of physiological function, reflecting biochemical, neurological, and structural changes. Through vocal analysis, it becomes possible to identify which system is breaking down—not just that a breakdown exists.
Methodology: Listening to Biology
Using BioAcoustic vocal profiling, three individuals diagnosed with Parkinson’s disease were evaluated. Each voice sample was analyzed across multiple biological layers, including:
Neurotransmitter function
Immune signaling
Detoxification pathways
Structural and muscular integrity
Despite sharing a common diagnosis, each individual demonstrated a distinct underlying pattern of dysfunction.
Case Study 1: The Gut–Immune–Neuroinflammation Pathway
Primary Features
Intestinal permeability (“leaky gut”)
Elevated inflammatory markers (Interleukin-17)
Tetanus-related bacterial signaling
Disruption of glutathione and glycine metabolism
Mechanism
In this case, the origin appears to begin in the gut:
Breakdown of the intestinal barrier ? immune activation ? systemic inflammation ? neuroinflammation ? motor symptoms
Interleukin-17, a pro-inflammatory cytokine, is known to promote neuroinflammation and disrupt the blood-brain barrier. Combined with impaired detoxification (glutathione dysfunction), this creates a biochemical environment that may damage dopaminergic neurons.
Clinical Implication
This type of Parkinson’s may be driven primarily by:
Immune dysregulation
Gut integrity failure
Chronic inflammatory signaling
Case Study 2- Thom: The Toxic–Oxidative Stress Pathway
Primary Features
Severe systemic toxicity
Widespread glutathione pathway disruption
Oxidative stress overload
Impaired cellular energy production
Mechanism
This profile reflects a breakdown in the body’s ability to detoxify:
Glutathione, a critical antioxidant, is central to neutralizing free radicals. In this case, multiple enzymes responsible for glutathione synthesis and utilization were compromised, indicating a system-wide failure of detoxification.
Clinical Implication
This type of Parkinson’s may be driven primarily by:
Environmental or metabolic toxicity
Antioxidant depletion
Mitochondrial dysfunction
Case Study 3- John: The Neurotransmitter–Structural Breakdown Pathway
Primary Features
Acetylcholine receptor dysfunction
Glycine receptor disruption
Impaired dopaminergic neuron support (GDNF pathway)
Muscle structure and nerve repair deficits
Mechanism
Here, the issue is not primarily immune or toxic, but neurological signaling and repair:
Neurotransmitter receptor dysfunction ? impaired signaling ? reduced neuron support ? failed repair mechanisms ? motor dysfunction
The GDNF (Glial Cell Line-Derived Neurotrophic Factor) pathway, essential for the survival of dopaminergic neurons, appears compromised. Simultaneously, structural proteins responsible for muscle coordination and nerve integrity show dysfunction.
Clinical Implication
This type of Parkinson’s may be driven primarily by:
Neurotransmitter imbalance
Failure of neuronal support systems
Structural degeneration of muscle and nerve tissue
Comparative Analysis: One Disease, Three Origins
These three cases reveal a critical insight:
Pathway
Primary Driver
Key System
Gut–Immune
Inflammation (IL-17)
Immune + Digestive
Toxic–Oxidative
Glutathione failure
Detox + Mitochondria
Neurotransmitter– Structural
Signaling breakdown
Nervous + Muscular
Despite identical diagnoses, the root causes differ dramatically.
Discussion: Rethinking Parkinson’s
The current medical model treats Parkinson’s as a uniform condition, often focusing on dopamine replacement. While this may address symptoms, it does not address cause.
These findings suggest:
Parkinson’s is a multi-origin condition
Symptoms are shared endpoints, not shared beginnings
Effective intervention must be pathway-specific
BioAcoustic analysis offers a unique advantage: it identifies dysfunction at the level of pattern and frequency, before structural damage becomes irreversible.
Conclusion: A New Model of Understanding
Parkinson’s disease may not be a single disease—but rather a collection of biological failures that sound the same.
If this is true, then:
Diagnosis must evolve beyond symptom classification
Treatment must become individualized
Early detection must focus on functional change, not structural damage
The human voice may provide a key to this shift—offering a non-invasive, real-time window into the body’s internal state.
Author Perspective
For over four decades, BioAcoustic Biology has explored the relationship between sound, structure, and physiology. The voice is not merely a tool for communication—it is a mathematical expression of the body’s current condition.
By learning to interpret this expression, we may move toward a future where disease is identified and addressed before it fully manifests.
COMPARATIVE SUMMARY (VERY IMPORTANT)
Mechanism
Client 1
Client 2
Client 3
Gut involvement
Strong
Immune / IL-17
Strong
Mild
Possible
Toxicity
Moderate
Severe
Secondary
Glutathione
(dominant)
Neurotransmitter receptors
Strong
Dopamine neuron support (GDNF)
Strong
Structural/muscle degeneration
Strong
“We won’t have a cure for diseases until we first have a cure for greed.”
What is the meaning of numbers on fruit stickers? Is that lemon you are thinking of buying at the grocery store really organic?
Numbers on fruit stickers at grocery stores have either 4 or 5 digits. If the fruit you are about to place into your grocery cart has a 4-digit code, might I suggest you leave the fruit on the grocery shelves. Four digit stickers mean that the fruit was grown using pesticides, fertilizers and other toxic chemicals.
If the code on your fruit is a 5-digit number, there are two possibilities. One is good and the other is not. If the 5-digit code begins with a 9, it shows that the produce was grown organically. Great, That is the preferred option.
If the code begins with an 8, it means that the fruit has been genetically modified. Best to pass on that option given widespread concerns about the dangers of genetically modified foods which have been banned in Europe. Over 19 countries, including France, Germany, and Italy have banned the cultivation of GMO crops
Now a caution is in order. Even if the fruit was grown organically – no harmful toxins applied – there still may be an accumulation of toxins from the soil.
The best option of all is to buy foods with no stickers from local farmers. Visit your local farmers market often. Befriend local farmers who grow their crops organically. You will enjoy the experience and best of all you will begin to feel better too.
A final note on the impact of stickers on food. The study below finds that young people do not make choices based on labeling whether a food was genetically modified or not. I recommend that everyone – young and old – take the labeling on food stickers into consideration when making purchase decisions.
Int J Environ Res Public Health. 2021 Feb 11;18(4):1761. GMO Food Labels Do Not Affect College Student Food Selection, Despite Negative Attitudes towards GMOs
Abstract
US Public Law 114-216 dictates that food producers in the United States of America will be required to label foods containing genetically modified organisms (GMOs) starting in 2022; however, there is little empirical evidence demonstrating how U.S. consumers would use food labels that indicate the presence or absence of GMOs.
The aim of this two-phase study was to determine how attitudes towards GMOs relate to food choices and how labels indicating the presence or absence of GMOs differentially impact choices among college students-the age group which values transparent food labeling more than any other. Participants (n = 434) made yes/no choices for each of 64 foods. In both phases of the study, participants were randomly assigned to seeing GMO Free labels, contains GMOs labels, or no GMO labels.
Across the two phases, 85% of participants reported believing that GMOs were at least somewhat dangerous to health (42% believed GMOs to be dangerous), yet in both studies, although eye-tracking data verified that participants attended to the GMO labels, these labels did not significantly affect food choices. Although college consumers may believe GMOs to be dangerous, their food choices do not reflect this belief.